Tumor angiogenesis is enforced by autocrine regulation of high-mobility group box 1. (Q45158260)

10 December 2017

Tumor angiogenesis is enforced by autocrine regulation of high-mobility group box 1. (English)

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10 December 2017

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J R van Beijnum

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10 December 2017

P Nowak-Sliwinska

2

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10 December 2017

E van den Boezem

3

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10 December 2017

P Hautvast

4

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10 December 2017

W A Buurman

5

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10 December 2017

A W Griffioen

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10 December 2017

language of work or name

English

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publication date

5 March 2012

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10 December 2017

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32

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363-374

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3

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https://scigraph.springernature.com/pub.10.1038/onc.2012.49

10 December 2017

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High-mobility group box 1 and cancer

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The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

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Receptor for advanced glycation end products (RAGE)-mediated neurite outgrowth and activation of NF-kappaB require the cytoplasmic domain of the receptor but different downstream signaling pathways

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HMGB1 interacts differentially with members of the Rel family of transcription factors

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C-reactive protein induces high-mobility group box-1 protein release through activation of p38MAPK in macrophage RAW264.7 cells.

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Role of HMGB1 in cardiovascular diseases

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Activation of Human Umbilical Vein Endothelial Cells Leads to Relocation and Release of High-Mobility Group Box Chromosomal Protein 1

31 March 2020

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Redox modification of cysteine residues regulates the cytokine activity of high mobility group box-1 (HMGB1)

31 March 2020

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Inhibition of tumor angiogenesis by HMGB1 A box peptide

31 March 2020

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Angiostatic kinase inhibitors to sustain photodynamic angio-occlusion

31 March 2020

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31 March 2020

Identifiers

DOI

10.1038/ONC.2012.49

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Dimensions Publication ID

10 December 2017

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