Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates. (Q45296599)

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scientific article published in May 2005

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English	Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates.	scientific article published in May 2005

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10 January 2020

Decreased expression of hypothalamic neuropeptides in Huntington disease transgenic mice with expanded polyglutamine-EGFP fluorescent aggregates (English)

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10 January 2020

Huntington's disease

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author name string

Svetlana Kotliarova

1

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Naoaki Sakamoto

3

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Masaru Kurosawa

4

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Haruko Miyazaki

5

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Munenori Nekooki

6

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Hiroshi Doi

7

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Yoko Machida

8

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Taishi Suzuki

10

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Chiharu Uchikawa

11

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Yuri Kotliarov

12

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Kazuyo Uchida

13

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Yoshiro Nagao

14

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Akira Tamaoka

16

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Kiyomitsu Oyanagi

17

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Nobuyuki Nukina

19

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publication date

1 May 2005

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number of pages

13

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Journal of Neurochemistry

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93

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3

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641-653

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10 January 2020

CART: from gene to function

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Aberrant interactions of transcriptional repressor proteins with the Huntington's disease gene product, huntingtin

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Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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Suberoylanilide hydroxamic acid, a histone deacetylase inhibitor, ameliorates motor deficits in a mouse model of Huntington's disease

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A YAC mouse model for Huntington's disease with full-length mutant huntingtin, cytoplasmic toxicity, and selective striatal neurodegeneration.

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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Age-dependent and tissue-specific CAG repeat instability occurs in mouse knock-in for a mutant Huntington's disease gene

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The hypothalamic lateral tuberal nucleus and the characteristics of neuronal loss in Huntington's disease

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Huntington's disease gene: regional and cellular expression in brain of normal and affected individuals

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Amino-terminal fragments of mutant huntingtin show selective accumulation in striatal neurons and synaptic toxicity

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Decreased expression of striatal signaling genes in a mouse model of Huntington's disease

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Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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Progressive formation of inclusions in the striatum and hippocampus of mice transgenic for the human Huntington's disease mutation

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Pivotal role of oligomerization in expanded polyglutamine neurodegenerative disorders.

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Huntingtin acts in the nucleus to induce apoptosis but death does not correlate with the formation of intranuclear inclusions.

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Expanded polyglutamine stretches interact with TAFII130, interfering with CREB-dependent transcription

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Trehalose alleviates polyglutamine-mediated pathology in a mouse model of Huntington disease

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