Mitochondrial-dependent Ca2+ handling in Huntington's disease striatal cells: effect of histone deacetylase inhibitors. (Q45302980)

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21 December 2019

title

Mitochondrial-dependent Ca2+ handling in Huntington's disease striatal cells: effect of histone deacetylase inhibitors (English)

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21 December 2019

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5

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21 December 2019

Michael R. Hayden

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Ana Cristina Rego

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Lisa M Ellerby

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21 December 2019

author name string

Jorge M A Oliveira

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21 December 2019

Sylvia Chen

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Sandra Almeida

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21 December 2019

Rebeccah Riley

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21 December 2019

David G Nicholls

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Catarina R Oliveira

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language of work or name

English

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publication date

1 October 2006

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21 December 2019

published in

Journal of Neuroscience

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volume

26

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21 December 2019

issue

43

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page(s)

11174-11186

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describes a project that uses

21 December 2019

ImageJ

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11 June 2022

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inferred from PubMed Central ID database lookup

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Enhanced Akt signaling is an early pro-survival response that reflects N-methyl-D-aspartate receptor activation in Huntington's disease knock-in striatal cells

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Histone Deacetylase Inhibition by Sodium Butyrate Chemotherapy Ameliorates the Neurodegenerative Phenotype in Huntington's Disease Mice

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12 December 2020

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12 December 2020

Striatal cells from mutant huntingtin knock-in mice are selectively vulnerable to mitochondrial complex II inhibitor-induced cell death through a non-apoptotic pathway

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12 December 2020

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Inhibition of mitochondrial complex II alters striatal expression of genes involved in glutamatergic and dopaminergic signaling: possible implications for Huntington's disease

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Potentiation of NMDA receptor-mediated excitotoxicity linked with intrinsic apoptotic pathway in YAC transgenic mouse model of Huntington's disease

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A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington's disease chromosomes.

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12 December 2020

10.1523/JNEUROSCI.3004-06.2006

Identifiers

DOI

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