GLP-1 signaling preserves cardiac function in endotoxemic Fischer 344 and DPP4-deficient rats. (Q45568816)

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14 February 2020

title

GLP-1 signaling preserves cardiac function in endotoxemic Fischer 344 and DPP4-deficient rats (English)

1 reference

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14 February 2020

2

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14 February 2020

Ming-Jai Su

series ordinal

3

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14 February 2020

author name string

Hui-Chun Ku

series ordinal

1

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publication date

18 September 2010

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Naunyn-Schmiedeberg's Archives of Pharmacology

14 February 2020

published in

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14 February 2020

volume

382

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issue

5-6

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14 February 2020

page(s)

463-474

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https://scigraph.springernature.com/pub.10.1007/s00210-010-0559-9

14 February 2020

exact match

0 references

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Glucagon-like peptide (GLP)-1(9-36)amide-mediated cytoprotection is blocked by exendin(9-39) yet does not require the known GLP-1 receptor.

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High-dose folic acid pretreatment blunts cardiac dysfunction during ischemia coupled to maintenance of high-energy phosphates and reduces postreperfusion injury

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Decreased dipeptidyl peptidase-IV activity and glucagon-like peptide-1(7-36)amide degradation in type 2 diabetic subjects.

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Somatostatin-28 regulates GLP-1 secretion via somatostatin receptor subtype 5 in rat intestinal cultures

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Active metabolite of GLP-1 mediates myocardial glucose uptake and improves left ventricular performance in conscious dogs with dilated cardiomyopathy

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Glucagon-like peptide-1 inhibits LPS-induced IL-1beta production in cultured rat astrocytes.

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