Transcription factor binding and histone modifications on the integrated proviral promoter in human T-cell leukemia virus-I-infected T-cells. (Q45730437)

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scientific article published on 16 October 2002

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English	Transcription factor binding and histone modifications on the integrated proviral promoter in human T-cell leukemia virus-I-infected T-cells.	scientific article published on 16 October 2002

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14 December 2019

Transcription factor binding and histone modifications on the integrated proviral promoter in human T-cell leukemia virus-I-infected T-cells (English)

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T-cell leukemia

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Nicholas J. Polakowski

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Isabelle Lemasson

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Paul J Laybourn

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Jennifer K Nyborg

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16 October 2002

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Journal of Biological Chemistry

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14 December 2019

49459-49465

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Isolation and characterization of retrovirus from cell lines of human adult T-cell leukemia and its implication in the disease

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Host sequences flanking the human T-cell leukemia virus type 1 provirus in vivo

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Clonal expansion of infected cells: a way of life for HTLV-I.

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Human T cell leukemia virus type I and neurologic disease: events in bone marrow, peripheral blood, and central nervous system during normal immune surveillance and neuroinflammation

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Location of cis-acting regulatory sequences in the human T-cell leukemia virus type I long terminal repeat

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Multiple sequence elements are required for regulation of human T-cell leukemia virus gene expression

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Human T-cell leukemia virus type 1 Tax requires direct access to DNA for recruitment of CREB binding protein to the viral promoter

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Specific regions of contact between human T-cell leukemia virus type I Tax protein and DNA identified by photocross-linking

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The human T-cell leukemia virus-1 transcriptional activator Tax enhances cAMP-responsive element-binding protein (CREB) binding activity through interactions with the DNA minor groove

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Human T-cell lymphotropic virus type I (HTLV-I) transcriptional activator, Tax, enhances CREB binding to HTLV-I 21-base-pair repeats by protein-protein interaction

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Complex formation between CREB and Tax enhances the binding affinity of CREB for the human T-cell leukemia virus type 1 21-base-pair repeats

1 reference

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Anchoring of CREB binding protein to the human T-cell leukemia virus type 1 promoter: a molecular mechanism of Tax transactivation

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Control of cAMP-regulated enhancers by the viral transactivator Tax through CREB and the co-activator CBP.

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Differential activation of viral and cellular promoters by human T-cell lymphotropic virus-1 tax and cAMP-responsive element modulator isoforms

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An exposed KID-like domain in human T-cell lymphotropic virus type 1 Tax is responsible for the recruitment of coactivators CBP/p300

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The coactivator CBP stimulates human T-cell lymphotrophic virus type I Tax transactivation in vitro

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The transcriptional coactivators p300 and CBP are histone acetyltransferases

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Molecular biology. Chromatin higher order folding--wrapping up transcription

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p300-mediated tax transactivation from recombinant chromatin: histone tail deletion mimics coactivator function

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Acetylation of nucleosomal histones by p300 facilitates transcription from tax-responsive human T-cell leukemia virus type 1 chromatin template

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The interaction of HTLV-1 Tax with HDAC1 negatively regulates the viral gene expression

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Functional interaction of the HTLV-1 transactivator Tax with activating transcription factor-4 (ATF4)

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Molecular interactions involved in the transactivation of the human T-cell leukemia virus type 1 promoter mediated by Tax and CREB-2 (ATF-4).

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Genetic characterization of transactivation of the human T-cell leukemia virus type 1 promoter: Binding of Tax to Tax-responsive element 1 is mediated by the cyclic AMP-responsive members of the CREB/ATF family of transcription factors

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The trans-activator tax of human T-cell leukemia virus type 1 (HTLV-1) interacts with cAMP-responsive element (CRE) binding and CRE modulator proteins that bind to the 21-base-pair enhancer of HTLV-1.

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Induction of the HTLV-I LTR by Jun occurs through the Tax-responsive 21-bp elements

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c-Jun, c-Fos and their family members activate the transcription mediated by three 21-bp repetitive sequences in the HTLV-I long terminal repeat.

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Human T-cell leukemia virus type 1 tax protein activates transcription through AP-1 site by inducing DNA binding activity in T cells

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In vivo genomic footprinting of the human T-cell leukemia virus type 1 (HTLV-1) long terminal repeat enhancer sequences in HTLV-1-infected human T-cell lines with different levels of Tax I activity

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10.1074/JBC.M209566200

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14 December 2019

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