Diabetes induced by Coxsackie virus: initiation by bystander damage and not molecular mimicry (Q45754853)

15 December 2017

title

Diabetes induced by Coxsackie virus: initiation by bystander damage and not molecular mimicry (English)

1 reference

15 December 2017

author name string

Horwitz MS

1

1 reference

15 December 2017

Bradley LM

2

1 reference

15 December 2017

Harbertson J

3

1 reference

15 December 2017

Krahl T

4

1 reference

15 December 2017

Lee J

5

1 reference

15 December 2017

Sarvetnick N

6

1 reference

15 December 2017

language of work or name

English

0 references

publication date

1 July 1998

1 reference

15 December 2017

published in

Nature Medicine

1 reference

15 December 2017

volume

4

1 reference

15 December 2017

page(s)

781-785

1 reference

15 December 2017

issue

7

1 reference

15 December 2017

https://scigraph.springernature.com/pub.10.1038/nm0798-781

exact match

0 references

cites work

Induction of bystander T cell proliferation by viruses and type I interferon in vivo

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Cross-reactivities in memory cytotoxic T lymphocyte recognition of heterologous viruses.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

High frequency of cross-reactive cytotoxic T lymphocytes elicited during the virus-induced polyclonal cytotoxic T lymphocyte response

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Viral infection of transgenic mice expressing a viral protein in oligodendrocytes leads to chronic central nervous system autoimmune disease

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Detection of coxsackie B virus RNA sequences in whole blood samples from adult patients at the onset of type I diabetes mellitus

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

A prospective study of the role of coxsackie B and other enterovirus infections in the pathogenesis of IDDM. Childhood Diabetes in Finland (DiMe) Study Group

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Virus-induced diabetes mellitus. XV. Beta cell damage and insulin-dependent hyperglycemia in mice infected with coxsackie virus B4

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Susceptibility of mice to group B coxsackie virus is influenced by the diabetic gene

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Diversity within a human isolate of coxsackie B4: Relationship to viral-induced diabetes

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Pathogenesis of virus-induced diabetes in mice

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Cellular immunity to a determinant common to glutamate decarboxylase and coxsackie virus in insulin-dependent diabetes

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Autoimmunity to two forms of glutamate decarboxylase in insulin-dependent diabetes mellitus

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

T cell cross-reactivity between coxsackievirus and glutamate decarboxylase is associated with a murine diabetes susceptibility allele

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Polygenic control of autoimmune diabetes in nonobese diabetic mice.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Following a diabetogenic T cell from genesis through pathogenesis

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Mechanism underlying counterregulation of autoimmune diabetes by IL-4.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Spontaneous loss of T-cell tolerance to glutamic acid decarboxylase in murine insulin-dependent diabetes

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

T helper cell subsets in insulin-dependent diabetes

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Viruses and T cell turnover: evidence for bystander proliferation

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Major expansion of CD8+ T cells with a predominant V beta usage during the primary immune response to HIV.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Longitudinal analysis of T cell receptor (TCR) gene usage by human immunodeficiency virus 1 envelope-specific cytotoxic T lymphocyte clones reveals a limited TCR repertoire

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

In vivo longitudinal analysis of a dominant TCR repertoire selected in human response to influenza virus

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Skewed T-cell receptor repertoire in genetically identical twins correlates with multiple sclerosis

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Evidence for superantigen involvement in insulin-dependent diabetes mellitus aetiology

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Massive expansion of antigen-specific CD8+ T cells during an acute virus infection

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Counting antigen-specific CD8 T cells: a reevaluation of bystander activation during viral infection.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Bystander activation of cytotoxic T cells: studies on the mechanism and evaluation of in vivo significance in a transgenic mouse model

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Minimal bystander activation of CD8 T cells during the virus-induced polyclonal T cell response

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Immune response to glutamic acid decarboxylase correlates with insulitis in non-obese diabetic mice.

1 reference

https://api.crossref.org/works/10.1038%2FNM0798-781

7 January 2021

Identifiers

DOI

10.1038/NM0798-781

1 reference

15 December 2017

Dimensions Publication ID

0 references

1 reference

15 December 2017