B-MYB delays cell aging by repressing p16 (INK4α) transcription. (Q45909407)

17 December 2017

title

B-MYB delays cell aging by repressing p16 (INK4α) transcription. (English)

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17 December 2017

author name string

Yu Huang

1

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17 December 2017

Junfeng Wu

2

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17 December 2017

Renzhong Li

3

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17 December 2017

Peichang Wang

4

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17 December 2017

Limin Han

5

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17 December 2017

Zongyu Zhang

6

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17 December 2017

Tanjun Tong

7

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17 December 2017

publication date

25 August 2010

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17 December 2017

Cellular and Molecular Life Sciences

published in

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17 December 2017

volume

68

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17 December 2017

issue

5

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17 December 2017

page(s)

893-901

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17 December 2017

https://scigraph.springernature.com/pub.10.1007/s00018-010-0501-9

exact match

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cites work

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Sp1 is essential for p16 expression in human diploid fibroblasts during senescence

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Cell aging of human diploid fibroblasts is associated with changes in responsiveness to epidermal growth factor and changes in HER-2 expression

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B-MYB, a transcription factor implicated in regulating cell cycle, apoptosis and cancer

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The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus

21 January 2018

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The Polycomb group proteins bind throughout the INK4A-ARF locus and are disassociated in senescent cells

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12 December 2020

Senescence delay of human diploid fibroblast induced by anti-sense p16INK4a expression

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12 December 2020

Stem-cell ageing modified by the cyclin-dependent kinase inhibitor p16INK4a

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12 December 2020

Transcriptional regulation of the CDK inhibitor p16INK4a gene by a novel pRb-associated repressor, RBAR1.

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12 December 2020

B-Myb protein in cellular proliferation, transcription control, and cancer: latest developments

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12 December 2020

10.1007/S00018-010-0501-9

Identifiers

DOI

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17 December 2017

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17 December 2017