Phosphorylation of connexin-43 at serine 262 promotes a cardiac injury-resistant state. (Q46021204)

18 December 2017

title

Phosphorylation of connexin-43 at serine 262 promotes a cardiac injury-resistant state. (English)

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18 December 2017

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6

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author name string

Wattamon Srisakuldee

1

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18 December 2017

Maya M Jeyaraman

2

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18 December 2017

Barbara E Nickel

3

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18 December 2017

Stéphane Tanguy

4

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18 December 2017

Zhi-Sheng Jiang

5

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18 December 2017

publication date

7 May 2009

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18 December 2017

published in

Cardiovascular Research

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18 December 2017

volume

83

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18 December 2017

issue

4

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18 December 2017

page(s)

672-681

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18 December 2017

Physiology of cardiovascular gap junctions

cites work

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https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Remodelling of gap junctions and connexin expression in heart disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Selective monoclonal antibody recognition and cellular localization of an unphosphorylated form of connexin43.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Temporal regulation of connexin phosphorylation in embryonic and adult tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Fibroblast growth factor-2 and cardioprotection

21 January 2018

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High- but not low-molecular weight FGF-2 causes cardiac hypertrophy in vivo; possible involvement of cardiotrophin-1.

21 January 2018

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Non-angiogenic FGF-2 protects the ischemic heart from injury, in the presence or absence of reperfusion

21 January 2018

1 reference

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Acute protection of ischemic heart by FGF-2: involvement of FGF-2 receptors and protein kinase C.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Phosphorylation of serine 262 in the gap junction protein connexin-43 regulates DNA synthesis in cell-cell contact forming cardiomyocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Ischemia-induced dephosphorylation of cardiomyocyte connexin-43 is reduced by okadaic acid and calyculin A but not fostriecin

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Phosphorylation at S365 is a gatekeeper event that changes the structure of Cx43 and prevents down-regulation by PKC.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Protein kinase C-epsilon mediates phorbol ester-induced phosphorylation of connexin-43.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

PKC-dependent phosphorylation may regulate the ability of connexin43 to inhibit DNA synthesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Delta-opioid receptor activation before ischemia reduces gap junction permeability in ischemic myocardium by PKC-epsilon-mediated phosphorylation of connexin 43.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Key connexin 43 phosphorylation events regulate the gap junction life cycle

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Intramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPT

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

High molecular weight FGF-2 promotes postconditioning-like cardioprotection linked to activation of protein kinase C isoforms, as well as Akt and p70 S6 kinases. [corrected].

21 January 2018

1 reference

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Gap junction-mediated spread of cell injury and death during myocardial ischemia-reperfusion.

21 January 2018

1 reference

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Protective role of gap junctions in preconditioning against myocardial infarction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Impairment of diazoxide-induced formation of reactive oxygen species and loss of cardioprotection in connexin 43 deficient mice

21 January 2018

1 reference

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Connexin 43 in cardiomyocyte mitochondria and its increase by ischemic preconditioning

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Cardioprotective mechanisms of PKC isozyme-selective activators and inhibitors in the treatment of ischemia-reperfusion injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Phosphorylation of connexin43 on serine368 by protein kinase C regulates gap junctional communication

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Change in permeant size selectivity by phosphorylation of connexin 43 gap-junctional hemichannels by PKC.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Identification of ischemia-regulated phosphorylation sites in connexin43: A possible target for the antiarrhythmic peptide analogue rotigaptide (ZP123).

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Regulation of purified and reconstituted connexin 43 hemichannels by protein kinase C-mediated phosphorylation of Serine 368.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Role of connexin 43 in ischemic preconditioning does not involve intercellular communication through gap junctions.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Connexin 43 in ischemic pre- and postconditioning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Connexin43 and ischemic preconditioning.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

Inhibition of phosphatase activity enhances preconditioning and limits cell death in the ischemic/reperfused aged rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVP142

21 January 2018

Identifiers

DOI

10.1093/CVR/CVP142

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18 December 2017

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18 December 2017