Glycine inhibitory dysfunction induces a selectively dynamic, morphine-resistant, and neurokinin 1 receptor- independent mechanical allodynia. (Q46108378)

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scientific article published in February 2009

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English	Glycine inhibitory dysfunction induces a selectively dynamic, morphine-resistant, and neurokinin 1 receptor- independent mechanical allodynia.	scientific article published in February 2009

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22 January 2020

Glycine inhibitory dysfunction induces a selectively dynamic, morphine-resistant, and neurokinin 1 receptor- independent mechanical allodynia (English)

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22 January 2020

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22 January 2020

Radhouane Dallel

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22 January 2020

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Loïs S Miraucourt

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22 January 2020

Daniel L Voisin

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1 February 2009

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22 January 2020

Journal of Neuroscience

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22 January 2020

2519-2527

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22 January 2020

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22 January 2020

Cardiovascular and neuronal responses to head stimulation reflect central sensitization and cutaneous allodynia in a rat model of migraine.

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ERK is sequentially activated in neurons, microglia, and astrocytes by spinal nerve ligation and contributes to mechanical allodynia in this neuropathic pain model

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BDNF from microglia causes the shift in neuronal anion gradient underlying neuropathic pain

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Disinhibition opens the gate to pathological pain signaling in superficial neurokinin 1 receptor-expressing neurons in rat spinal cord.

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/19244526

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based on heuristic

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Hyperalgesia induced by altered glycinergic activity at the spinal cord

1 reference

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1 reference

https://pubmed.ncbi.nlm.nih.gov/19244526

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Receptor endocytosis and dendrite reshaping in spinal neurons after somatosensory stimulation

1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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1 reference

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Neuronal model of tactile allodynia produced by spinal strychnine: effects of excitatory amino acid receptor antagonists and a mu-opiate receptor agonist

1 reference

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Spinal effects of bicuculline: modulation of an allodynia-like state by an A1-receptor agonist, morphine, and an NMDA-receptor antagonist

1 reference

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Morphine administered in the substantia gelatinosa of the spinal trigeminal nucleus caudalis inhibits nociceptive activities in the spinal trigeminal nucleus oralis.

1 reference

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Differential effects of morphine on responses of dorsal horn lamina V type cells elicited by A and C fibre stimulation in the spinal cat

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Identifiers

10.1523/JNEUROSCI.3923-08.2009

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22 January 2020

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Europe PubMed Central

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22 January 2020

1 reference

Europe PubMed Central

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22 January 2020

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