Different pathways by which extracellular Ca2+ promotes calcitonin gene-related peptide release from central terminals of capsaicin-sensitive afferents of guinea pigs: effect of capsaicin, high K+ and low pH media (Q46332932)

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scientific article published on December 6, 1991

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English	Different pathways by which extracellular Ca2+ promotes calcitonin gene-related peptide release from central terminals of capsaicin-sensitive afferents of guinea pigs: effect of capsaicin, high K+ and low pH media	scientific article published on December 6, 1991

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scholarly article

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Europe PubMed Central

21 December 2017

http://europepmc.org/abstract/MED/1726064

Different pathways by which extracellular Ca2+ promotes calcitonin gene-related peptide release from central terminals of capsaicin-sensitive afferents of guinea pigs: effect of capsaicin, high K+ and low pH media (English)

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10.1016/0006-8993(91)91679-U

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9 November 2022

omega-conotoxins

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Europe PubMed Central

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9 November 2022

author name string

E. Del Bianco

1

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9 November 2022

P. Santicioli

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9 November 2022

M. Tramontana

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9 November 2022

C. A. Maggi

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9 November 2022

R. Cecconi

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9 November 2022

P. Geppetti

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9 November 2022

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publication date

1 December 1991

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21 December 2017

http://europepmc.org/abstract/MED/1726064

6 December 1991

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9 November 2022

full work available at URL

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9 November 2022

https://api.elsevier.com/content/article/PII:000689939191679U?httpAccept=text/plain

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9 November 2022

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21 December 2017

http://europepmc.org/abstract/MED/1726064

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http://europepmc.org/abstract/MED/1726064

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21 December 2017

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46-53

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21 December 2017

http://europepmc.org/abstract/MED/1726064

Ruthenium red selectively inhibits capsaicin-induced release of calcitonin gene-related peptide from the isolated perfused guinea pig lung

1

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Ruthenium red selectively prevents capsaicin-induced nociceptor stimulation

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Protons activate a cation conductance in a sub-population of rat dorsal root ganglion neurones

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Capsaicin-evoked neuropeptide release is not dependent on membrane potential changes

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Ruthenium red blocks the capsaicin-induced increase in intracellular calcium and activation of membrane currents in sensory neurones as well as the activation of peripheral nociceptors in vitro.

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Low pH-induced release of calcitonin gene-related peptide from capsaicin-sensitive sensory nerves: Mechanism of action and biological response

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Distribution of calcitonin gene-related peptide-like immunoreactivity in various rat tissues: correlation with substance P and other tachykinins and sensitivity to capsaicin

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Thiorphan increases capsaicin-evoked release of substance P from slices of dorsal spinal cord of guinea pig.

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Neurochemical evidence for the activation of the 'efferent' function of capsaicin-sensitive nerves by lowering of the pH in the guinea-pig urinary bladder.

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Characterization of the electrically evoked release of substance P from dorsal root ganglion neurons: methods and dihydropyridine sensitivity.

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Local effector functions of capsaicin-sensitive sensory nerve endings: involvement of tachykinins, calcitonin gene-related peptide and other neuropeptides

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Proton-induced transformation of calcium channel in chick dorsal root ganglion cells

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A receptor for protons in the nerve cell membrane

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Protective action of ruthenium red toward capsaicin desensitization of sensory fibers

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The effect of omega conotoxin GVIA, a peptide modulator of the N-type voltage sensitive calcium channels, on motor responses produced by activation of efferent and sensory nerves in mammalian smooth muscle

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The effect of calcium free medium and nifedipine on the release of substance P-like immunoreactivity and contractions induced by capsaicin in the isolated guinea-pig and rat bladder.

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Calcitonin gene-related peptide coexists with substance P in capsaicin sensitive neurons and sensory ganglia of the rat.

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10.1016/0006-8993(91)91679-U

1 reference

Europe PubMed Central

21 December 2017

http://europepmc.org/abstract/MED/1726064

1 reference

Europe PubMed Central

21 December 2017

http://europepmc.org/abstract/MED/1726064

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