TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells. (Q46344187)

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scientific article published on 26 September 2008

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English	TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells.	scientific article published on 26 September 2008

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scholarly article

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14 January 2020

TLR4/MYD88-dependent, LPS-induced synthesis of PGE2 by macrophages or dendritic cells prevents anti-CD3-mediated CD95L upregulation in T cells (English)

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14 January 2020

toll-like receptor

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Karina Bortoluci

2

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14 January 2020

Ricardo Weinlich

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14 January 2020

Carlos Henrique Serezani

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C F Chehab

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A G Ulbrich

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M Peters-Golden

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M Russo

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G P Amarante-Mendes

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26 September 2008

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Cell Death & Differentiation

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1901-1909

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14 January 2020

https://scigraph.springernature.com/pub.10.1038/cdd.2008.128

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Signal transduction by the lipopolysaccharide receptor, Toll-like receptor-4

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Prostaglandins as modulators of immunity

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Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

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Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

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Peptide-stimulated DO11.10 T cells divide well but accumulate poorly in the absence of TLR agonist treatment

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