Different mechanisms of saturated versus polyunsaturated FFA-induced apoptosis in human endothelial cells (Q46444254)

22 December 2017

title

Different mechanisms of saturated versus polyunsaturated FFA-induced apoptosis in human endothelial cells (English)

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22 December 2017

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8

Michael Wolzt

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22 December 2017

author name string

Michaela Artwohl

1

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22 December 2017

Andrea Lindenmair

2

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22 December 2017

Veronika Sexl

3

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22 December 2017

Christina Maier

4

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22 December 2017

Georg Rainer

5

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22 December 2017

Angelika Freudenthaler

6

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22 December 2017

Nicole Huttary

7

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22 December 2017

Peter Nowotny

9

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22 December 2017

Anton Luger

10

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22 December 2017

Sabina M Baumgartner-Parzer

11

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22 December 2017

publication date

5 August 2008

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22 December 2017

Journal of Lipid Research

published in

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22 December 2017

volume

49

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22 December 2017

issue

12

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22 December 2017

page(s)

2627-2640

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22 December 2017

Free fatty acids are associated with obesity, insulin resistance, and atherosclerosis in renal transplant recipients.

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Unsaturated fatty acids isolated from human lipoproteins activate protein phosphatase type 2Cbeta and induce apoptosis in endothelial cells

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Fatty acids liberated from low-density lipoprotein trigger endothelial apoptosis via mitogen-activated protein kinases

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Saturated, but not unsaturated, fatty acids induce apoptosis of human coronary artery endothelial cells via nuclear factor-kappaB activation.

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Endothelial cell apoptosis: biochemical characteristics and potential implications for atherosclerosis

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Number and migratory activity of circulating endothelial progenitor cells inversely correlate with risk factors for coronary artery disease

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p38 mitogen-activated protein kinase mediates palmitate-induced apoptosis but not inhibitor of nuclear factor-kappaB degradation in human coronary artery endothelial cells

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Lipolysis products from triglyceride-rich lipoproteins increase endothelial permeability, perturb zonula occludens-1 and F-actin, and induce apoptosis

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c-Myc primed mitochondria determine cellular sensitivity to TRAIL-induced apoptosis

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Caspase-8 can be activated by interchain proteolysis without receptor-triggered dimerization during drug-induced apoptosis.

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XRCC1 is specifically associated with poly(ADP-ribose) polymerase and negatively regulates its activity following DNA damage

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Base excision repair proteins are required for integrin-mediated suppression of bleomycin-induced DNA breakage in murine lung endothelial cells.

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21 January 2018

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10.1194/JLR.M800393-JLR200

21 January 2018

Identifiers

DOI

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22 December 2017

PubMed ID

18682607

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22 December 2017