Activated protein C prevents inflammation yet stimulates angiogenesis to promote cutaneous wound healing. (Q46543534)

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12 January 2020

title

Activated protein C prevents inflammation yet stimulates angiogenesis to promote cutaneous wound healing (English)

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12 January 2020

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1 reference

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Christopher J Jackson

1

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12 January 2020

author name string

Meilang Xue

2

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12 January 2020

Patrick Thompson

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12 January 2020

Ross A Davey

4

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12 January 2020

Kaley Whitmont

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Susan Smith

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Nathalie Buisson-Legendre

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Tamara Sztynda

8

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Louise J Furphy

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Alan Cooper

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12 January 2020

Philip Sambrook

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Lyn March

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12 January 2020

publication date

1 May 2005

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Wound Repair and Regeneration

12 January 2020

published in

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volume

13

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12 January 2020

issue

3

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12 January 2020

page(s)

284-294

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12 January 2020

cites work

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Recombinant human activated protein C attenuates the inflammatory response in endothelium and monocytes by modulating nuclear factor-kappaB.

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Activated protein C inhibits lipopolysaccharide-induced nuclear translocation of nuclear factor kappaB (NF-kappaB) and tumour necrosis factor alpha (TNF-alpha) production in the THP-1 monocytic cell line

21 January 2018

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Gene expression profile of antithrombotic protein c defines new mechanisms modulating inflammation and apoptosis

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Activated protein C blocks p53-mediated apoptosis in ischemic human brain endothelium and is neuroprotective

21 January 2018

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Activated protein C: potential therapy for severe sepsis, thrombosis, and stroke

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Topical vascular endothelial growth factor accelerates diabetic wound healing through increased angiogenesis and by mobilizing and recruiting bone marrow-derived cells

21 January 2018

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Bioactive interleukin-8 is expressed in wounds and enhances wound healing

21 January 2018

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Local overexpression of monocyte chemoattractant protein-1 at vessel wall induces infiltration of macrophages and formation of atherosclerotic lesion: synergism with hypercholesterolemia

21 January 2018

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21 January 2018

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Activated protein C induces endothelial cell proliferation by mitogen-activated protein kinase activation in vitro and angiogenesis in vivo.

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