Selenoprotein deficiency and high levels of selenium compounds can effectively inhibit hepatocarcinogenesis in transgenic mice. (Q46709847)

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7 January 2020

title

Selenoprotein deficiency and high levels of selenium compounds can effectively inhibit hepatocarcinogenesis in transgenic mice (English)

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7 January 2020

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1

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7 January 2020

Vyacheslav Labunskyy

3

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7 January 2020

Mohamed E. Moustafa

7

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9

Vadim N Gladyshev

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7 January 2020

author name string

Diego F Calvisi

2

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7 January 2020

Valentina M Factor

4

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Bradley A Carlson

5

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Dmitri E Fomenko

6

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Dolph L Hatfield

8

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language of work or name

English

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publication date

1 December 2005

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7 January 2020

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7 January 2020

volume

24

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issue

54

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7 January 2020

page(s)

8003-8011

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https://scigraph.springernature.com/pub.10.1038/sj.onc.1208940

7 January 2020

exact match

0 references

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Disruption of beta-catenin pathway or genomic instability define two distinct categories of liver cancer in transgenic mice

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Specific excision of the selenocysteine tRNA[Ser]Sec (Trsp) gene in mouse liver demonstrates an essential role of selenoproteins in liver function

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Disruption of redox homeostasis in the transforming growth factor-alpha/c-myc transgenic mouse model of accelerated hepatocarcinogenesis.

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Vitamin E reduces chromosomal damage and inhibits hepatic tumor formation in a transgenic mouse model

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Selenium deficiency abrogates inflammation-dependent plasma cell tumors in mice

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Opposite roles of selenium-dependent glutathione peroxidase-1 in superoxide generator diquat- and peroxynitrite-induced apoptosis and signaling

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Contrasting patterns of regulation of the antioxidant selenoproteins, thioredoxin reductase, and glutathione peroxidase, in cancer cells

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7 January 2021

Protective role of selenium against hepatitis B virus and primary liver cancer in Qidong

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7 January 2021

Diverse Effects of Methylseleninic Acid on the Transcriptional Program of Human Prostate Cancer Cells

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7 January 2021