Cleavage of epidermal growth factor receptor by caspase during apoptosis is independent of its internalization (Q46772694)

25 December 2017

title

Cleavage of epidermal growth factor receptor by caspase during apoptosis is independent of its internalization (English)

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25 December 2017

3

C F Chignell

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25 December 2017

author name string

Y-Y He

series ordinal

1

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25 December 2017

J-L Huang

series ordinal

2

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language of work or name

English

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publication date

1 March 2006

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25 December 2017

published in

Oncogene

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25 December 2017

volume

25

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issue

10

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page(s)

1521-1531

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25 December 2017

https://scigraph.springernature.com/pub.10.1038/sj.onc.1209184

exact match

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cites work

Genetic deficiency or pharmacological inhibition of cyclooxygenase-1 or -2 induces mouse keratinocyte differentiation in vitro and in vivo

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Proteolytic cleavage of epidermal growth factor receptor by caspases.

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Caspase-8-dependent HER-2 cleavage in response to tumor necrosis factor alpha stimulation is counteracted by nuclear factor kappaB through c-FLIP-L expression

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Developmental cell death: morphological diversity and multiple mechanisms

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Membrane blebbing during apoptosis results from caspase-mediated activation of ROCK I.

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Regulated portals of entry into the cell

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Endocytosis and mitogenic signaling

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Mammalian caspases: structure, activation, substrates, and functions during apoptosis

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Delayed and sustained activation of extracellular signal-regulated kinase in human keratinocytes by UVA: implications in carcinogenesis

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Epidermal growth factor receptor down-regulation induced by UVA in human keratinocytes does not require the receptor kinase activity.

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Role of reduced glutathione efflux in apoptosis of immortalized human keratinocytes induced by UVA.

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Desensitisation of cultured glial cells to epidermal growth factor by receptor down-regulation

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Gefitinib--a novel targeted approach to treating cancer

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Apoptosis: a link between cancer genetics and chemotherapy

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A functional role for death proteases in s-Myc- and c-Myc-mediated apoptosis

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Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

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Distinct signaling pathways are involved in leukosialin (CD43) down-regulation, membrane blebbing, and phospholipid scrambling during neutrophil apoptosis

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Disruption of mitochondrial function during apoptosis is mediated by caspase cleavage of the p75 subunit of complex I of the electron transport chain

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Extraction of cholesterol with methyl-beta-cyclodextrin perturbs formation of clathrin-coated endocytic vesicles

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The erbB family: targets for therapeutic development against cancer and therapeutic strategies using monoclonal antibodies and tyrosine kinase inhibitors

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Internally quenched fluorescent peptide substrates disclose the subsite preferences of human caspases 1, 3, 6, 7 and 8

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Acute cholesterol depletion inhibits clathrin-coated pit budding

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Apoptosis in the pathogenesis and treatment of disease

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Proapoptotic function of the MET tyrosine kinase receptor through caspase cleavage

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Signal transduction by receptors with tyrosine kinase activity

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Cell death: the significance of apoptosis

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Downregulation of epidermal growth factor receptor signaling by singlet oxygen through activation of caspase-3 and protein phosphatases

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1209184

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25 December 2017

Dimensions Publication ID

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25 December 2017