Treating lithium-induced nephrogenic diabetes insipidus with a COX-2 inhibitor improves polyuria via upregulation of AQP2 and NKCC2. (Q46794013)

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1 January 2020

title

Treating lithium-induced nephrogenic diabetes insipidus with a COX-2 inhibitor improves polyuria via upregulation of AQP2 and NKCC2 (English)

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nephrogenic diabetes insipidus

1 January 2020

main subject

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1

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1 January 2020

Ju Young Jung

3

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1 January 2020

7

Mark A Knepper

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1 January 2020

author name string

Nak Won Choi

2

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1 January 2020

Ji-Hyun Song

4

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1 January 2020

Chang Hwa Lee

5

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1 January 2020

Chong Myung Kang

6

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publication date

23 January 2008

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American Journal of Physiology - Renal Physiology

1 January 2020

published in

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volume

294

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1 January 2020

page(s)

F702-9

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1 January 2020

issue

4

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Evidence for an in vivo antagonism between vasopressin and prostaglandin in the mammalian kidney

1 January 2020

cites work

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Prevalence, pathogenesis, and treatment of renal dysfunction associated with chronic lithium therapy

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Pathogenesis of nephrogenic diabetes insipidus due to chronic administration of lithium in rats

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On the mechanism of lithium-induced diabetes insipidus in man and the rat

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Effects of specific inhibition of cyclo-oxygenase-1 and cyclo-oxygenase-2 in the rat stomach with normal mucosa and after acid challenge

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Vasopressin-prostaglandin interactions in the regulation of epithelial cell permeability to water

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Normal hemodynamic and coagulation responses to 1-deamino-8-D-arginine vasopressin in a case of lithium-induced nephrogenic diabetes insipidus. Results of treatment by a prostaglandin synthesis inhibitor (indomethacin)

21 January 2018

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Antidiuretic effect of hydrochlorothiazide in lithium-induced nephrogenic diabetes insipidus is associated with upregulation of aquaporin-2, Na-Cl co-transporter, and epithelial sodium channel

21 January 2018

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Chlorpropamide action on renal concentrating mechanism in rats with hypothalamic diabetes insipidus

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Development of lithium-induced nephrogenic diabetes insipidus is dissociated from adenylyl cyclase activity.

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In vivo effect of indomethacin to potentiate the renal medullary cyclic AMP response to vasopressin

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Treatment of congenital nephrogenic diabetes insipidus by hydrochlorothiazide and cyclooxygenase-2 inhibitor

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Lithium treatment inhibits renal GSK-3 activity and promotes cyclooxygenase 2-dependent polyuria

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