Dok1 mediates high-fat diet-induced adipocyte hypertrophy and obesity through modulation of PPAR-gamma phosphorylation. (Q46801706)

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1 January 2020

title

Dok1 mediates high-fat diet-induced adipocyte hypertrophy and obesity through modulation of PPAR-gamma phosphorylation (English)

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6

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Mashito Sakai

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1 January 2020

author name string

Tetsuya Hosooka

1

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1 January 2020

Tetsuya Noguchi

2

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1 January 2020

Ko Kotani

3

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1 January 2020

Takehiro Nakamura

4

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1 January 2020

Hiroshi Sakaue

5

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Wataru Ogawa

7

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1 January 2020

Kazutoshi Tobimatsu

8

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1 January 2020

Kazuo Takazawa

9

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1 January 2020

Yasushi Matsuki

11

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1 January 2020

Ryuji Hiramatsu

12

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1 January 2020

Tomoharu Yasuda

13

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1 January 2020

Mitchell A Lazar

14

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Yuji Yamanashi

15

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1 January 2020

Masato Kasuga

16

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language of work or name

English

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publication date

20 January 2008

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1 January 2020

volume

14

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issue

2

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1 January 2020

page(s)

188-193

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Insulin signaling in health and disease

1 January 2020

cites work

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7 January 2021

Increased Insulin Sensitivity and Hypoinsulinemia in APS Knockout Mice

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Disruption of the SH2-B gene causes age-dependent insulin resistance and glucose intolerance

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c-Cbl-deficient mice have reduced adiposity, higher energy expenditure, and improved peripheral insulin action

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Deletion of Gab1 in the liver leads to enhanced glucose tolerance and improved hepatic insulin action

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p62(dok): a constitutively tyrosine-phosphorylated, GAP-associated protein in chronic myelogenous leukemia progenitor cells

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Identification of the Abl- and rasGAP-associated 62 kDa protein as a docking protein, Dok

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p62(dok), a negative regulator of Ras and mitogen-activated protein kinase (MAPK) activity, opposes leukemogenesis by p210(bcr-abl)

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7 January 2021

Insulin receptor-mediated p62dok tyrosine phosphorylation at residues 362 and 398 plays distinct roles for binding GTPase-activating protein and Nck and is essential for inhibiting insulin-stimulated activation of Ras and Akt.

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7 January 2021

Tyrosine phosphorylation of p62(Dok) induced by cell adhesion and insulin: possible role in cell migration

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Inhibition of the motility and growth of B16F10 mouse melanoma cells by dominant negative mutants of Dok-1.

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7 January 2021

Molecular regulation of adipogenesis

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Transcriptional regulation of metabolism

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7 January 2021

Dok-1 and Dok-2 are negative regulators of lipopolysaccharide-induced signaling

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7 January 2021

PPAR gamma mediates high-fat diet-induced adipocyte hypertrophy and insulin resistance

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7 January 2021

Improved insulin-sensitivity in mice heterozygous for PPAR-gamma deficiency

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7 January 2021

Compensation by the muscle limits the metabolic consequences of lipodystrophy in PPAR gamma hypomorphic mice

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7 January 2021

Adipose-specific peroxisome proliferator-activated receptor gamma knockout causes insulin resistance in fat and liver but not in muscle

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7 January 2021

Deletion of PPARgamma in adipose tissues of mice protects against high fat diet-induced obesity and insulin resistance

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7 January 2021

Inhibition of adipogenesis through MAP kinase-mediated phosphorylation of PPARgamma

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7 January 2021

Transcriptional activation by peroxisome proliferator-activated receptor gamma is inhibited by phosphorylation at a consensus mitogen-activated protein kinase site

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7 January 2021

Regulation of peroxisome proliferator-activated receptor gamma activity by mitogen-activated protein kinase

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7 January 2021

Interdomain communication regulating ligand binding by PPAR-gamma

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7 January 2021

Genetic modulation of PPARgamma phosphorylation regulates insulin sensitivity

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7 January 2021

Peroxisome proliferator-activated receptor-gamma: too much of a good thing causes harm

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7 January 2021

Adipose tissue selective insulin receptor knockout protects against obesity and obesity-related glucose intolerance

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Skp2 controls adipocyte proliferation during the development of obesity.

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7 January 2021