Nitric oxide-cGMP-protein kinase G signaling pathway induces anoxic preconditioning through activation of ATP-sensitive K+ channels in rat hearts. (Q46844253)

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English	Nitric oxide-cGMP-protein kinase G signaling pathway induces anoxic preconditioning through activation of ATP-sensitive K+ channels in rat hearts.	scientific article

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10 January 2020

Nitric oxide-cGMP-protein kinase G signaling pathway induces anoxic preconditioning through activation of ATP-sensitive K+ channels in rat hearts (English)

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Jae Boum Youm

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Dang Van Cuong

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Nari Kim

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Hyun Joo

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Jae-Wha Lee

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Won Sun Park

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Taeho Kim

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Sunghyun Kang

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Hyungkyu Kim

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Jin Han

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9 December 2005

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American Journal of Physiology Heart and Circulatory Physiology

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H1808-17

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10 January 2020

Microvascular oxygenation, oxidative stress, NO suppression and superoxide dismutase during postischemic reperfusion.

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Inducible nitric oxide synthase in the myocard

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Mitochondrial K(ATP) channel opening protects a human atrial-derived cell line by a mechanism involving free radical generation

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The mitochondrial permeability transition pore

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Mechanisms by which opening the mitochondrial ATP- sensitive K(+) channel protects the ischemic heart

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Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

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Mechanism of potassium efflux and action potential shortening during ischaemia in isolated mammalian cardiac muscle

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The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene

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Blockade of the ATP-sensitive potassium channel by taurine in rabbit ventricular myocytes.

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ATP-sensitive K(+) channel activation by nitric oxide and protein kinase G in rabbit ventricular myocytes

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Modulation of ATP-sensitive potassium channels by cGMP-dependent protein kinase in rabbit ventricular myocytes

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K(ATP) channel-independent targets of diazoxide and 5-hydroxydecanoate in the heart

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Inhibiting mitochondrial permeability transition pore opening at reperfusion protects against ischaemia-reperfusion injury

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Diazoxide triggers cardioprotection against apoptosis induced by oxidative stress

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Opening of potassium channels: the common cardioprotective link between preconditioning and natural hibernation?

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Nitric oxide. Biochemistry, pathophysiology, and detection

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Oxidative stress and cardiac disease

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Mitochondrial ATP-dependent potassium channels: novel effectors of cardioprotection?

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Nitric oxide inhibits ischemia/reperfusion-induced myocardial apoptosis by modulating cyclin A-associated kinase activity

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The single cell gel electrophoresis assay (comet assay): a European review

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Does nitric oxide modulate mitochondrial energy generation and apoptosis?

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Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

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Ventricular assist device therapy normalizes inducible nitric oxide synthase expression and reduces cardiomyocyte apoptosis in the failing human heart.

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Sarcolemmal and mitochondrial K(ATP) channels and myocardial ischemic preconditioning.

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Activation of mitochondrial ATP-dependent potassium channels by nitric oxide.

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Modulation of mitochondrial ATP-dependent K+ channels by protein kinase C.

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Activation of distinct cAMP-dependent and cGMP-dependent pathways by nitric oxide in cardiac myocytes

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