Ethanol exposure and withdrawal sensitizes the rat hippocampal CA1 pyramidal cell region to beta-amyloid (25-35)-induced cytotoxicity: NMDA receptor involvement (Q46844787)

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English	Ethanol exposure and withdrawal sensitizes the rat hippocampal CA1 pyramidal cell region to beta-amyloid (25-35)-induced cytotoxicity: NMDA receptor involvement	scientific article

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Europe PubMed Central

PubMed publication ID

25 December 2017

http://europepmc.org/abstract/MED/16340465

Ethanol exposure and withdrawal sensitizes the rat hippocampal CA1 pyramidal cell region to beta-amyloid (25-35)-induced cytotoxicity: NMDA receptor involvement (English)

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Europe PubMed Central

PubMed publication ID

25 December 2017

http://europepmc.org/abstract/MED/16340465

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Mark A Prendergast

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Mark A Prendergast

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25 December 2017

http://europepmc.org/abstract/MED/16340465

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Rachel L Self

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25 December 2017

http://europepmc.org/abstract/MED/16340465

Katherine J Smith

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25 December 2017

http://europepmc.org/abstract/MED/16340465

Patrick J Mulholland

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25 December 2017

http://europepmc.org/abstract/MED/16340465

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1 November 2005

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25 December 2017

http://europepmc.org/abstract/MED/16340465

Alcoholism: Clinical and Experimental Research

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25 December 2017

http://europepmc.org/abstract/MED/16340465

29

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25 December 2017

http://europepmc.org/abstract/MED/16340465

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25 December 2017

http://europepmc.org/abstract/MED/16340465

2063-2069

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25 December 2017

http://europepmc.org/abstract/MED/16340465

Topographic distribution of neurofibrillary tangles and granulovacuolar degeneration in hippocampal cortex of aging and demented patients. A quantitative study

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Hydrogen peroxide mediates amyloid beta protein toxicity

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Beta-amyloid fragment 25-35 causes mitochondrial dysfunction in primary cortical neurons

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Ethanol sensitivity in ATP-gated P2X receptors is subunit dependent

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Polyamines contribute to ethanol withdrawal-induced neurotoxicity in rat hippocampal slice cultures through interactions with the NMDA receptor

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Ethanol withdrawal hyper‐responsiveness mediated by NMDA receptors in spinal cord motor neurons

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MGLuR5 activation reduces beta-amyloid-induced cell death in primary neuronal cultures and attenuates translocation of cytochrome c and apoptosis-inducing factor

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Increased sensitivity to NMDA is involved in alcohol-withdrawal induced cytotoxicity observed in primary cultures of cortical neurones chronically pre-treated with ethanol.

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Hippocampal CA1 region neurodegeneration produced by ethanol withdrawal requires activation of intrinsic polysynaptic hippocampal pathways and function of N-methyl-D-aspartate receptors

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Glutamate-induced increase of extracellular glutamate through N-methyl-D-aspartate receptors in ethanol withdrawal

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Chronic ethanol modulates delta and mu-opioid receptor expression in rat CNS: immunohistochemical analysis with quantitiative confocal microscopy

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Beta-amyloid peptide 25-35 regulates basal and hormone-stimulated Ca2+ levels in cultured rat astrocytes

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A slowly formed transient conformer of Abeta(1-40) is toxic to inward channels of dissociated hippocampal and cortical neurons of rats

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Amyloid beta-protein induced electrophysiological changes are dependent on aggregation state: N-methyl-D-aspartate (NMDA) versus non-NMDA receptor/channel activation

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Identifiers

10.1097/01.ALC.0000187591.82039.B2

1 reference

Europe PubMed Central

PubMed publication ID

25 December 2017

http://europepmc.org/abstract/MED/16340465

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

25 December 2017

http://europepmc.org/abstract/MED/16340465

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