Trans-generational exposure to low levels of rhodamine B does not adversely affect litter size or liver function in murine mucopolysaccharidosis type IIIA. (Q47422772)

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scientific article published on 22 June 2010

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English	Trans-generational exposure to low levels of rhodamine B does not adversely affect litter size or liver function in murine mucopolysaccharidosis type IIIA.	scientific article published on 22 June 2010

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8 February 2020

Trans-generational exposure to low levels of rhodamine B does not adversely affect litter size or liver function in murine mucopolysaccharidosis type IIIA (English)

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author name string

Ainslie L K Roberts

1

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Janice M Fletcher

2

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Lynette Moore

3

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22 June 2010

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Molecular Genetics and Metabolism

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2-3

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208-213

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Recombinant human sulphamidase: expression, amplification, purification and characterization

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Improvement in behaviour after substrate deprivation therapy with rhodamine B in a mouse model of MPS IIIA.

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Inhibition of glycosaminoglycan synthesis using rhodamine B in a mouse model of mucopolysaccharidosis type IIIA.

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Studies of the metabolism of rhodamine B

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Acute exposure to rhodamine B

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A mouse model for mucopolysaccharidosis type III A (Sanfilippo syndrome).

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Gastrointestinal pathology in a mouse model of mucopolysaccharidosis type IIIA

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New method for quantitative determination of uronic acids

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Genistein-mediated inhibition of glycosaminoglycan synthesis as a basis for gene expression-targeted isoflavone therapy for mucopolysaccharidoses

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Genistein reduces lysosomal storage in peripheral tissues of mucopolysaccharide IIIB mice

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Genistein: a natural isoflavone with a potential for treatment of genetic diseases

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Genistin-rich soy isoflavone extract in substrate reduction therapy for Sanfilippo syndrome: An open-label, pilot study in 10 pediatric patients

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Identifiers

10.1016/J.YMGME.2010.06.008

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8 February 2020

1 reference

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8 February 2020

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