Selective Direct Toxicity of Cocaine on Fetal Mouse Neurons: Teratogenic Implications of Neurite and Apoptotic Neuronal Loss (Q47671724)

4 February 2018

title

Selective Direct Toxicity of Cocaine on Fetal Mouse Neurons: Teratogenic Implications of Neurite and Apoptotic Neuronal Loss (English)

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4 February 2018

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Philippe Evrard

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4 February 2018

author name string

Marie-Cécile Nassogne

series ordinal

1

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4 February 2018

Pierre J Courtoy

series ordinal

3

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4 February 2018

language of work or name

English

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publication date

1 June 1998

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4 February 2018

Annals of the New York Academy of Sciences

published in

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4 February 2018

volume

846

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4 February 2018

issue

1

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4 February 2018

page(s)

51-68

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4 February 2018

The prevalence of illicit-drug or alcohol use during pregnancy and discrepancies in mandatory reporting in Pinellas County, Florida

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Risk of intracranial hemorrhage and other adverse outcomes after cocaine exposure in a cohort of 323 very low birth weight infants

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Cocaine does not compromise cerebral or myocardial oxygen delivery in fetal sheep.

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Prenatal cocaine exposure adversely affects development of the primate cerebral cortex.

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Prenatal cocaine delays astroglial maturation: immunodensitometry shows increased markers of immaturity (vimentin and GAP-43) and decreased proliferation and production of the growth factor S-100.

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Cocaine acutely inhibits DNA synthesis in developing rat brain regions: evidence for direct actions

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Postnatal age defines specificity of immediate early gene induction by cocaine in developing rat brain.

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Identifiers

DOI

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4 February 2018

release_gyh5yveddjgofot3qv4b3g6fea

Fatcat ID

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https://api.fatcat.wiki/v0/release/gyh5yveddjgofot3qv4b3g6fea

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4 February 2018