Oxytocin ameliorates the immediate myocardial injury in rat heart transplant through downregulation of neutrophil-dependent myocardial apoptosis. (Q47761332)

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English	Oxytocin ameliorates the immediate myocardial injury in rat heart transplant through downregulation of neutrophil-dependent myocardial apoptosis.	scientific article

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scholarly article

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

Oxytocin ameliorates the immediate myocardial injury in rat heart transplant through downregulation of neutrophil-dependent myocardial apoptosis. (English)

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

1 reference

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author name string

F Al-Amran

1

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

M Shahkolahi

2

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

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publication date

1 July 2013

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5 February 2018

http://europepmc.org/abstract/MED/23953571

Transplantation Proceedings

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

45

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

2506-2512

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Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

6

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

Negative inotropic and chronotropic effects of oxytocin

1 reference

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Neonatal oxytocin treatment modulates oxytocin receptor, atrial natriuretic peptide, nitric oxide synthase and estrogen receptor mRNAs expression in rat heart

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Oxytocin receptor genetic variation relates to empathy and stress reactivity in humans

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Plasma oxytocin is related to lower cardiovascular and sympathetic reactivity to stress

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Rat heart: a site of oxytocin production and action

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7 January 2021

Oxytocin releases atrial natriuretic peptide by combining with oxytocin receptors in the heart

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Oxytocin-Gly-Lys-Arg: a novel cardiomyogenic peptide

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Oxytocin protects rat heart against ischemia-reperfusion injury via pathway involving mitochondrial ATP-dependent potassium channel.

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Anti-inflammatory effect of oxytocin in rat myocardial infarction.

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Oxytocin attenuates NADPH-dependent superoxide activity and IL-6 secretion in macrophages and vascular cells

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C1q-mediated chemotaxis by human neutrophils: involvement of gClqR and G-protein signalling mechanisms

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Chemokines and chemokine receptors in leukocyte trafficking

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Neutrophil-activating peptide-1/interleukin 8, a novel cytokine that activates neutrophils

1 reference

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Single Administration of the CXC Chemokine-Binding Protein Evasin-3 During Ischemia Prevents Myocardial Reperfusion Injury in Mice

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Myocardium functional recovery protection by omeprazole after ischemia-reperfusion in isolated rat hearts

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Inside ST-elevation myocardial infarction by monitoring concentrations of cardiovascular risk biomarkers in blood

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Crosstalk between leptin and interleukin-1β abrogates negative inotropic effects in a model of chronic hyperleptinemia

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Exacerbation of myocardial injury in transgenic mice overexpressing FGF-2 is T cell dependent

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Neutrophil ablation with anti-serum does not protect against phase 2 ventricular arrhythmias in anaesthetised rats with myocardial infarction.

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Lactoperoxidase-catalyzed lipid peroxidation of microsomal and artificial membranes

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A novel tool for B-cell tolerance research: characterization of mouse alloantibody development using a simple and reliable cellular ELISA technique

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The innate immune response in reperfused myocardium

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Myocardial infarction induces early increased remote ADAM8 expression of rat hearts after cardiac arrest

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Lipocortin 1 and chemokine modulation of granulocyte and monocyte accumulation in experimental inflammation

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Dissociation of oxytocin, vasopressin and corticotropin secretion during different types of stress

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Reactive oxygen molecules, oxidant injury and renal disease

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Mechanisms of neutrophil-induced parenchymal cell injury

1 reference

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10.1016/J.TRANSPROCEED.2013.03.022

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

1 reference

Europe PubMed Central

5 February 2018

http://europepmc.org/abstract/MED/23953571

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