Association of UCP2 -866 G/A polymorphism with chronic inflammatory diseases (Q47959866)

7 February 2018

title

Association of UCP2 -866 G/A polymorphism with chronic inflammatory diseases (English)

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13

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Marta E Alarcón-Riquelme

14

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Saleh Ibrahim

16

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Tom H Karlsen

7

T H Karlsen

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Kirsten Muri Boberg

8

K M Boberg

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Annika Bergquist

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A Bergquist

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Andre Franke

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A Franke

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author name string

X Yu

1

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S Wieczorek

2

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H Yin

4

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M Pierer

5

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C Sina

6

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M Kunz

10

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T Witte

11

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W L Gross

12

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S Schreiber

15

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publication date

23 April 2009

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published in

Genes and Immunity

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volume

10

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issue

6

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page(s)

601-605

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https://scigraph.springernature.com/pub.10.1038/gene.2009.29

exact match

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Mouse models for mitochondrial disease

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mtDNA nt13708A variant increases the risk of multiple sclerosis

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A common polymorphism in the promoter of UCP2 is associated with decreased risk of obesity in middle-aged humans

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https://api.crossref.org/works/10.1038%2FGENE.2009.29

Uncoupling protein 2 promoter polymorphism -866G/A affects its expression in beta-cells and modulates clinical profiles of Japanese type 2 diabetic patients

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https://api.crossref.org/works/10.1038%2FGENE.2009.29

Disruption of the uncoupling protein-2 gene in mice reveals a role in immunity and reactive oxygen species production

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Retinoids activate proton transport by the uncoupling proteins UCP1 and UCP2.

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https://api.crossref.org/works/10.1038%2FGENE.2009.29

Uncoupling protein 2 has protective function during experimental autoimmune encephalomyelitis.

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Mitochondrial DNA modifies cognition in interaction with the nuclear genome and age in mice

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mt-Nd2 Allele of the ALR/Lt mouse confers resistance against both chemically induced and autoimmune diabetes

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Dissecting the effects of mtDNA variations on complex traits using mouse conplastic strains.

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Association of a common polymorphism in the promoter of UCP2 with susceptibility to multiple sclerosis

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inferred from DOI database lookup

7 January 2021

based on heuristic

Mitochondrial DNA mutations in multiple sclerosis

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7 January 2021

based on heuristic

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https://api.crossref.org/works/10.1038%2FGENE.2009.29

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1038/GENE.2009.29

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7 February 2018

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7 February 2018