Crohn's disease-associated Nod2 mutants reduce IL10 transcription. (Q47975511)

7 February 2018

title

Crohn's disease-associated Nod2 mutants reduce IL10 transcription. (English)

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7 February 2018

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Dana J Philpott

1

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7 February 2018

Stephen E Girardin

series ordinal

2

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7 February 2018

language of work or name

English

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publication date

1 May 2009

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7 February 2018

published in

Nature Immunology

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7 February 2018

volume

10

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7 February 2018

issue

5

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7 February 2018

page(s)

455-457

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7 February 2018

https://scigraph.springernature.com/pub.10.1038/ni0509-455

exact match

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cites work

Unravelling the pathogenesis of inflammatory bowel disease

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

A frameshift mutation in NOD2 associated with susceptibility to Crohn's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Predictive value of the CARD15 variant 1007fs for the diagnosis of intestinal stenoses and the need for surgery in Crohn's disease in clinical practice: results of a prospective study.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Nod2, a Nod1/Apaf-1 family member that is restricted to monocytes and activates NF-kappaB

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Function of Nod-like receptors in microbial recognition and host defense

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Nod2 is a general sensor of peptidoglycan through muramyl dipeptide (MDP) detection

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Host recognition of bacterial muramyl dipeptide mediated through NOD2. Implications for Crohn's disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

RICK/RIP2 Mediates Innate Immune Responses Induced through Nod1 and Nod2 but Not TLRs

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

The frameshift mutation in Nod2 results in unresponsiveness not only to Nod2- but also Nod1-activating peptidoglycan agonists

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Inflammatory bowel disease: clinical aspects and established and evolving therapies

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Faecalibacterium prausnitzii is an anti-inflammatory commensal bacterium identified by gut microbiota analysis of Crohn disease patients

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

Nod2 mutation in Crohn's disease potentiates NF-kappaB activity and IL-1beta processing

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FNI0509-455

A Crohn's disease-associated NOD2 mutation suppresses transcription of human IL10 by inhibiting activity of the nuclear ribonucleoprotein hnRNP-A1.

21 January 2018

1 reference

PubMed

https://pubmed.ncbi.nlm.nih.gov/19381138

inferred from PubMed ID database lookup

12 December 2020

based on heuristic

Identifiers

DOI

10.1038/NI0509-455

1 reference

7 February 2018

Dimensions Publication ID

1005501545

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Fatcat ID

release_hr76aa3gtndh7myrfsgjhv2ujm

0 references

PubMed ID

19381138

1 reference

7 February 2018