Alterations in G1/S cell-cycle control contributing to carcinogenesis. (Q48003436)

7 February 2018

review article

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title

Alterations in G1/S cell-cycle control contributing to carcinogenesis. (English)

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7 February 2018

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Kaelin WG Jr

1

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7 February 2018

language of work or name

English

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publication date

1 December 1997

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7 February 2018

Annals of the New York Academy of Sciences

published in

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7 February 2018

volume

833

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7 February 2018

page(s)

29-33

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7 February 2018

Cyclin D1 overexpression vs. retinoblastoma inactivation: implications for growth control evasion in non-small cell and small cell lung cancer

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DP and E2F proteins: components of a heterodimeric transcription factor implicated in cell cycle control

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DRTF1/E2F: an expanding family of heterodimeric transcription factors implicated in cell-cycle control

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E2F: a link between the Rb tumor suppressor protein and viral oncoproteins

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Inhibition of E2F-1 transactivation by direct binding of the retinoblastoma protein

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E2F-1-mediated transactivation is inhibited by complex formation with the retinoblastoma susceptibility gene product

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A Restriction Point for Control of Normal Animal Cell Proliferation

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In vitro phosphorylation of the tumor suppressor gene RB protein by mitosis-specific histone H1 kinase

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The E2F transcription factor is a cellular target for the RB protein

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Molecular cloning of cellular genes encoding retinoblastoma-associated proteins: identification of a gene with properties of the transcription factor E2F

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Functional interactions of the retinoblastoma protein with mammalian D-type cyclins

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Regulation of retinoblastoma protein functions by ectopic expression of human cyclins

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Retinoblastoma protein switches the E2F site from positive to negative element

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Mechanism of active transcriptional repression by the retinoblastoma protein

21 January 2018

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A potent transrepression domain in the retinoblastoma protein induces a cell cycle arrest when bound to E2F sites

21 January 2018

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The retinoblastoma susceptibility gene product represses transcription when directly bound to the promoter

21 January 2018

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Direct transcriptional repression by pRB and its reversal by specific cyclins

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The transcription factor E2F-1 is a downstream target of RB action

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RB [corrected] as a modulator of transcription

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Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis

21 January 2018

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Expression of transcription factor E2F1 induces quiescent cells to enter S phase

21 January 2018

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Oncogenic capacity of the E2F1 gene

21 January 2018

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Multiple members of the E2F transcription factor family are the products of oncogenes

21 January 2018

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https://api.crossref.org/works/10.1111%2FJ.1749-6632.1997.TB48589.X

Deregulated expression of E2F-1 induces S-phase entry and leads to apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1749-6632.1997.TB48589.X

p53 and E2F-1 cooperate to mediate apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1111%2FJ.1749-6632.1997.TB48589.X

10.1111/J.1749-6632.1997.TB48589.X

21 January 2018

Identifiers

DOI

1 reference

7 February 2018

PubMed ID

9616737

1 reference

7 February 2018