Tyrosine kinase A-nerve growth factor receptor is antigenically present in dystrophic neurites from a variety of conditions but not in Alzheimer's disease. (Q48101376)

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English	Tyrosine kinase A-nerve growth factor receptor is antigenically present in dystrophic neurites from a variety of conditions but not in Alzheimer's disease.	scientific article

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scholarly article

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31 October 2019

Tyrosine kinase A-nerve growth factor receptor is antigenically present in dystrophic neurites from a variety of conditions but not in Alzheimer's disease (English)

1 reference

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31 October 2019

Alzheimer's disease

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1 reference

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Lucio Marinelli

1

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31 October 2019

3

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31 October 2019

Giovanna Angelini

6

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31 October 2019

Massimo Tabaton

7

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M Tabaton

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31 October 2019

author name string

S Cammarata

2

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31 October 2019

A Schenone

4

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31 October 2019

D Zaccheo

5

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31 October 2019

language of work or name

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publication date

1 September 1999

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31 October 2019

number of pages

5

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Neuroscience Letters

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31 October 2019

273

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31 October 2019

1

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31 October 2019

67-71

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31 October 2019

Measurement of nerve growth factor-like immunoreactivity in human brain using an anti-mouse-NGF enzyme immunoassay

1 reference

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Dendritic Growth in the Aged Human Brain and Failure of Growth in Senile Dementia

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Selective loss of central cholinergic neurons in Alzheimer's disease

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Activation of K+ channels and suppression of neuronal activity by secreted beta-amyloid-precursor protein.

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Senile plaques as aberrant sprout-stimulating structures.

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Nerve growth factor mRNA in peripheral and central rat tissues and in the human central nervous system: lesion effects in the rat brain and levels in Alzheimer's disease.

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The contribution of altered synapses in the senile plaque: an electron microscopic study in Alzheimer's dementia.

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Neurotrophin signalling

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Regulated neurotrophin receptor responsiveness during neuronal migrationand early differentiation.

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Neurotrophic factors: from molecule to man.

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Neurotrophins and brain insults.

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TrkA activation is sufficient to rescue axotomized cholinergic neurons

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Nerve growth factor increases mRNA levels for the prion protein and the beta-amyloid protein precursor in developing hamster brain

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Reduction in p140-TrkA receptor protein within the nucleus basalis and cortex in Alzheimer's disease

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Expression of high- and low-affinity neurotrophin receptors on human transformed B lymphocytes

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The widespread alteration of neurites in Alzheimer's disease may be unrelated to amyloid deposition

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Alzheimer disease: Evidence for selective loss of cholinergic neurons in the nucleus basalis

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Identifiers

10.1016/S0304-3940(99)00625-4

1 reference

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31 October 2019

1 reference

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31 October 2019

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