Neuroprotection by ATP-dependent potassium channels in rat neocortical brain slices during hypoxia (Q48101455)

8 February 2018

title

Neuroprotection by ATP-dependent potassium channels in rat neocortical brain slices during hypoxia (English)

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8 February 2018

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S Garcia de Arriba

1

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8 February 2018

H Franke

2

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8 February 2018

M Pissarek

3

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8 February 2018

K Nieber

4

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8 February 2018

P Illes

5

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8 February 2018

language of work or name

English

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publication date

1 September 1999

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8 February 2018

published in

Neuroscience Letters

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8 February 2018

volume

273

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issue

1

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8 February 2018

page(s)

13-16

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8 February 2018

Properties and functions of ATP-sensitive K-channels

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Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

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The high-affinity sulphonylurea receptor regulates KATP channels in nerve terminals of the rat motor cortex

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Changes by short-term hypoxia in the membrane properties of pyramidal cells and the levels of purine and pyrimidine nucleotides in slices of rat neocortex; effects of agonists and antagonists of ATP-dependent potassium channels.

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Activation of ATP-sensitive potassium channels decreases neuronal injury caused by chemical hypoxia.

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Whole-cell recording of anoxic effects on hippocampal neurons in slices.

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Effect of potassium channel modulators on the release of glutamate induced by ischaemic-like conditions in rat hippocampal slices.

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Identifiers

DOI

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8 February 2018

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8 February 2018