Hypoxic preconditioning induces elevated expression of stanniocalcin-1 in the heart. (Q48134076)

9 February 2018

title

Hypoxic preconditioning induces elevated expression of stanniocalcin-1 in the heart. (English)

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9 February 2018

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Johan A Westberg

1

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9 February 2018

Martina Serlachius

series ordinal

2

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9 February 2018

Petri Lankila

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3

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9 February 2018

Leif C Andersson

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4

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publication date

15 June 2007

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9 February 2018

American Journal of Physiology Heart and Circulatory Physiology

published in

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9 February 2018

volume

293

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9 February 2018

issue

3

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9 February 2018

page(s)

H1766-71

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9 February 2018

Normobaric hypoxia induces tolerance to focal permanent cerebral ischemia in association with an increased expression of hypoxia-inducible factor-1 and its target genes, erythropoietin and VEGF, in the adult mouse brain.

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The respiratory effects of stanniocalcin-1 (STC-1) on intact mitochondria and cells: STC-1 uncouples oxidative phosphorylation and its actions are modulated by nucleotide triphosphates

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Delayed effects of sublethal ischemia on the acquisition of tolerance to ischemia

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Cardiac stress protein elevation 24 hours after brief ischemia or heat stress is associated with resistance to myocardial infarction

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Characterization of mammalian stanniocalcin receptors. Mitochondrial targeting of ligand and receptor for regulation of cellular metabolism

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Preconditioning with ischemia: a delay of lethal cell injury in ischemic myocardium

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Stanniocalcin-1 is a naturally occurring L-channel inhibitor in cardiomyocytes: relevance to human heart failure

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Purification, characterization, and bioassay of teleocalcin, a glycoprotein from salmon corpuscles of Stannius

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Hypoxic preconditioning induces neuroprotective stanniocalcin-1 in brain via IL-6 signaling.

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Nuclear factor interleukin 6 motifs mediate tissue-specific gene transcription in hypoxia

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Hypoxia-inducible factor-1-mediated activation of stanniocalcin-1 in human cancer cells.

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Stanniocalcin 1 as a pleiotropic factor in mammals.

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Whole-body hypoxic preconditioning protects mice against acute hypoxia by improving lung function.

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10.1152/AJPHEART.00017.2007

21 January 2018

Identifiers

DOI

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9 February 2018

PubMed ID

17573464

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9 February 2018