Sodium salicylate protects against rotenone-induced parkinsonism in rats. (Q48145970)

9 February 2018

title

Sodium salicylate protects against rotenone-induced parkinsonism in rats. (English)

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9 February 2018

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parkinsonian syndrome

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Sindhu K Madathil

1

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9 February 2018

Saravanan S Karuppagounder

series ordinal

2

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9 February 2018

Kochupurackal P Mohanakumar

series ordinal

3

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publication date

27 March 2013

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9 February 2018

published in

Synapse

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volume

67

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issue

8

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page(s)

502-514

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Nonsteroidal anti-inflammatory drugs in Parkinson's disease: possible involvement of quinone formation

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Loss of mitochondrial complex I activity potentiates dopamine neuron death induced by microtubule dysfunction in a Parkinson's disease model

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Quercetin up-regulates mitochondrial complex-I activity to protect against programmed cell death in rotenone model of Parkinson's disease in rats

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Combining nitric oxide release with anti-inflammatory activity preserves nigrostriatal dopaminergic innervation and prevents motor impairment in a 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model of Parkinson's disease

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Switching the microglial harmful phenotype promotes lifelong restoration of subtantia nigra dopaminergic neurons from inflammatory neurodegeneration in aged mice

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Nitric oxide synthase inhibitors protect against rotenone-induced, oxidative stress mediated parkinsonism in rats.

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Acetaminophen and aspirin inhibit superoxide anion generation and lipid peroxidation, and protect against 1-methyl-4-phenyl pyridinium-induced dopaminergic neurotoxicity in rats

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Non-steroidal anti-inflammatory drug sodium salicylate, but not diclofenac or celecoxib, protects against 1-methyl-4-phenyl pyridinium-induced dopaminergic neurotoxicity in rats

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L-deprenyl protects against rotenone-induced, oxidative stress-mediated dopaminergic neurodegeneration in rats

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Acute intranigral infusion of rotenone in rats causes progressive biochemical lesions in the striatum similar to Parkinson's disease

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Melatonin protects against rotenone-induced oxidative stress in a hemiparkinsonian rat model.

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Ability to delay neuropathological events associated with astrocytic MAO-B increase in a Parkinsonian mouse model: implications for early intervention on disease progression.

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Behavioral differences in a rotenone-induced hemiparkinsonian rat model developed following intranigral or median forebrain bundle infusion

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Rats with unilateral median forebrain bundle, but not striatal or nigral, lesions by the neurotoxins MPP+ or rotenone display differential sensitivity to amphetamine and apomorphine.

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Nonsteroidal anti-inflammatory drugs may protect against Parkinson disease

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Identification and quantitation of glutathione in hepatic protein mixed disulfides and its relationship to glutathione disulfide

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Identifiers

DOI

10.1002/SYN.21658

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9 February 2018

PubMed ID

23447126

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9 February 2018