Activation of nuclear factor kappaB by nitric oxide in rat striatal neurones: differential inhibition of the p50 and p65 subunits by dexamethasone. (Q48174963)

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English	Activation of nuclear factor kappaB by nitric oxide in rat striatal neurones: differential inhibition of the p50 and p65 subunits by dexamethasone.	scientific article

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

Activation of nuclear factor kappaB by nitric oxide in rat striatal neurones: differential inhibition of the p50 and p65 subunits by dexamethasone. (English)

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

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C S Simpson

1

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

B J Morris

2

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

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1 July 1999

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

Journal of Neurochemistry

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

73

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

1

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

353-361

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Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

Activation of DNA-binding activity in an apparently cytoplasmic precursor of the NF-kappa B transcription factor

1 reference

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Function and activation of NF-kappa B in the immune system

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NF-kappa B-like factors in the murine brain. Developmentally-regulated and tissue-specific expression

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Global ischemia activates nuclear factor-kappa B in forebrain neurons of rats.

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Tumor necrosis factor alpha activates human immunodeficiency virus type 1 through induction of nuclear factor binding to the NF-kappa B sites in the long terminal repeat

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Transcription factor NF-kappaB is activated in primary neurons by amyloid beta peptides and in neurons surrounding early plaques from patients with Alzheimer disease

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Constitutive NF-kappa B activity in neurons

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Stimulation of ionotropic glutamate receptors activates transcription factor NF-kappa B in primary neurons

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Nuclear factor-kappa B mediates induction of vascular cell adhesion molecule-1 in glomerular mesangial cells.

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Brain Aging Correlates: Retardation by Hormonal-Pharmacological Treatments

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HTLV-1 transactivator induces interleukin-2 receptor expression through an NF-kappa B-like factor.

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Non-classical glutamate receptors, blocked by both NMDA and non-NMDA antagonists, stimulate nitric oxide production in neurons.

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Inhibition of NF-kappaB DNA binding by nitric oxide

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Steroids and depression

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Evidence for involvement of NF-kappaB in the transcriptional control of COX-2 gene expression by IL-1beta

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NF-kappa B: a crucial transcription factor for glial and neuronal cell function

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Induction and stabilization of I kappa B alpha by nitric oxide mediates inhibition of NF-kappa B.

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Induction of inhibitory factor kappaBalpha mRNA in the central nervous system after peripheral lipopolysaccharide administration: an in situ hybridization histochemistry study in the rat

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Nuclear factor kappa B activates proenkephalin transcription in T lymphocytes

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Physical association and functional antagonism between the p65 subunit of transcription factor NF-kappa B and the glucocorticoid receptor

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Lipopolysaccharide-induced nitric oxide synthase activity in cultured cerebellar granule neurons.

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Involvement of transcriptional mechanisms in the inhibition of NOS2 expression by dexamethasone in rat mesangial cells.

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Differential regulation of vascular cell adhesion molecule 1 gene expression by specific NF-kappa B subunits in endothelial and epithelial cells

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Induction of c-fos and zif/268 gene expression in rat striatal neurons, following stimulation of D1-like dopamine receptors, involves protein kinase A and protein kinase C.

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Neuronal (type I) nitric oxide synthase regulates nuclear factor kappaB activity and immunologic (type II) nitric oxide synthase expression

1 reference

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Co-stimulatory effect of nitric oxide on endothelial NF-kappaB implies a physiological self-amplifying mechanism

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Inhibition of NFkB DNA binding activity by glucocorticoids in rat brain

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Distinct domains of the RelA NF-kappaB subunit are required for negative cross-talk and direct interaction with the glucocorticoid receptor.

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Identifiers

10.1046/J.1471-4159.1999.0730353.X

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

1 reference

Europe PubMed Central

9 February 2018

http://europepmc.org/abstract/MED/10386988

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