Reduced tissue damage and improved recovery of motor function after traumatic brain injury in mice deficient in complement component C4. (Q48189458)

9 February 2018

title

Reduced tissue damage and improved recovery of motor function after traumatic brain injury in mice deficient in complement component C4. (English)

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9 February 2018

author name string

Zerong You

1

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9 February 2018

Jinsheng Yang

2

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9 February 2018

Kazue Takahashi

3

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9 February 2018

Phoebe H Yager

4

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9 February 2018

Hyung-Hwan Kim

5

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9 February 2018

Tao Qin

6

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9 February 2018

Gregory L Stahl

7

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9 February 2018

R Alan B Ezekowitz

8

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9 February 2018

Michael C Carroll

9

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9 February 2018

Michael J Whalen

10

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9 February 2018

publication date

25 April 2007

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9 February 2018

Journal of Cerebral Blood Flow & Metabolism

published in

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9 February 2018

volume

27

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9 February 2018

issue

12

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page(s)

1954-1964

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9 February 2018

Complement activation in the human brain after traumatic head injury

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Region-selective effects of neuroinflammation and antioxidant treatment on peripheral benzodiazepine receptors and NMDA receptors in the rat brain

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Dynamic changes in N-methyl-D-aspartate receptors after closed head injury in mice: Implications for treatment of neurological and cognitive deficits

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Complement C4 inhibits systemic autoimmunity through a mechanism independent of complement receptors CR1 and CR2

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Caspase-3 mediated neuronal death after traumatic brain injury in rats

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boc-Aspartyl(OMe)-fluoromethylketone attenuates mitochondrial release of cytochrome c and delays brain tissue loss after traumatic brain injury in rats.

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In stroke, complement will get you nowhere

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A murine model for neuropsychiatric disorders associated with group A beta-hemolytic streptococcal infection.

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Effect of soluble complement receptor-1 on neutrophil accumulation after traumatic brain injury in rats

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Activated complement components C3a and C4a in cerebrospinal fluid and plasma following subarachnoid hemorrhage

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Local neutrophil influx following lateral fluid-percussion brain injury in rats is associated with accumulation of complement activation fragments of the third component (C3) of the complement system

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Elevated levels of the complement components C3 and factor B in ventricular cerebrospinal fluid of patients with traumatic brain injury.

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Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

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Pharmacological complement inhibition at the C3 convertase level promotes neuronal survival, neuroprotective intracerebral gene expression, and neurological outcome after traumatic brain injury.

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Central nervous system-targeted complement inhibition mediates neuroprotection after closed head injury in transgenic mice

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Closed head injury--an inflammatory disease?

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Complement activated C4d immunoreactive oligodendrocytes delineate small cortical plaques in multiple sclerosis.

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Inducible nitric oxide synthase is an endogenous neuroprotectant after traumatic brain injury in rats and mice

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Intracerebral complement C5a receptor (CD88) expression is regulated by TNF and lymphotoxin-alpha following closed head injury in mice

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7 January 2021

Intrathecal levels of complement-derived soluble membrane attack complex (sC5b-9) correlate with blood-brain barrier dysfunction in patients with traumatic brain injury.

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7 January 2021

A targeted disruption of the murine complement factor B gene resulting in loss of expression of three genes in close proximity, factor B, C2, and D17H6S45.

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7 January 2021

C1q-deficiency is neuroprotective against hypoxic-ischemic brain injury in neonatal mice

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7 January 2021

Glial responses, clusterin, and complement in permanent focal cerebral ischemia in the mouse

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7 January 2021

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Genomic responses in rat cerebral cortex after traumatic brain injury

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Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complement

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Studies of group B streptococcal infection in mice deficient in complement component C3 or C4 demonstrate an essential role for complement in both innate and acquired immunity

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7 January 2021

Reduced brain edema after traumatic brain injury in mice deficient in P-selectin and intercellular adhesion molecule-1

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7 January 2021

Intestinal reperfusion injury is mediated by IgM and complement

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7 January 2021

The role of complement C3 in intracerebral hemorrhage-induced brain injury.

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7 January 2021

Predominant role for C5b-9 in renal ischemia/reperfusion injury.

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7 January 2021

Identifiers

DOI

10.1038/SJ.JCBFM.9600497

1 reference

9 February 2018

release_4wphy2htxfgjzn7zu4bujsbvbq

Fatcat ID

0 references

1 reference

9 February 2018