Novel monoclonal antibodies demonstrate biochemical variation of brain parkin with age. (Q48199015)

9 February 2018

title

Novel monoclonal antibodies demonstrate biochemical variation of brain parkin with age. (English)

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Ted M Dawson

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John Q Trojanowski

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author name string

Aaron C Pawlyk

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Benoit I Giasson

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Deepak M Sampathu

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Francisco A Perez

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Kah Leong Lim

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Valina L Dawson

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Richard D Palmiter

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Virginia M-Y Lee

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language of work or name

English

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publication date

12 September 2003

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Journal of Biological Chemistry

published in

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volume

278

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issue

48

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page(s)

48120-48128

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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

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Parkin functions as an E2-dependent ubiquitin- protein ligase and promotes the degradation of the synaptic vesicle-associated protein, CDCrel-1

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Parkin localizes to the Lewy bodies of Parkinson disease and dementia with Lewy bodies

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Immunohistochemical and subcellular localization of Parkin protein: absence of protein in autosomal recessive juvenile parkinsonism patients

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Parkin immunoreactivity in the brain of human and non-human primates: an immunohistochemical analysis in normal conditions and in Parkinsonian syndromes

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Neurodegeneration with brain iron accumulation, type 1 is characterized by alpha-, beta-, and gamma-synuclein neuropathology

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Lewy bodies contain altered alpha-synuclein in brains of many familial Alzheimer's disease patients with mutations in presenilin and amyloid precursor protein genes

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21 January 2018

Identifiers

DOI

10.1074/JBC.M306889200

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9 February 2018

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9 February 2018