GABA and histogenesis in fetal and neonatal mouse brain lacking both the isoforms of glutamic acid decarboxylase. (Q48231628)

9 February 2018

title

GABA and histogenesis in fetal and neonatal mouse brain lacking both the isoforms of glutamic acid decarboxylase. (English)

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9 February 2018

author name string

Ji F

series ordinal

1

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9 February 2018

Kanbara N

series ordinal

2

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9 February 2018

Obata K

series ordinal

3

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9 February 2018

publication date

1 March 1999

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9 February 2018

published in

Neuroscience Research

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9 February 2018

volume

33

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9 February 2018

issue

3

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9 February 2018

page(s)

187-194

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9 February 2018

Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures

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Cleft palate and decreased brain gamma-aminobutyric acid in mice lacking the 67-kDa isoform of glutamic acid decarboxylase

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Involvement of GABAA receptors in the outgrowth of cultured hippocampal neurons.

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GABA stimulates chemotaxis and chemokinesis of embryonic cortical neurons via calcium-dependent mechanisms.

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Differential response of cortical plate and ventricular zone cells to GABA as a migration stimulus.

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GABAA, NMDA and AMPA receptors: a developmentally regulated 'ménage à trois'.

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Molecular genetic analysis of synaptic plasticity, activity-dependent neural development, learning, and memory in the mammalian brain

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7 January 2021

Targeting of the 67-kDa isoform of glutamic acid decarboxylase to intracellular organelles is mediated by its interaction with the NH2-terminal region of the 65-kDa isoform of glutamic acid decarboxylase

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Mapping of glutamic acid decarboxylase (GAD) genes

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A ventrodorsal GABA gradient in the embryonic retina prior to expression of glutamate decarboxylase

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Two genes encode distinct glutamate decarboxylases

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Comparative localization of mRNAs encoding two forms of glutamic acid decarboxylase with nonradioactive in situ hybridization methods

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Comparative localization of two forms of glutamic acid decarboxylase and their mRNAs in rat brain supports the concept of functional differences between the forms

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GABA inhibits migration of luteinizing hormone-releasing hormone neurons in embryonic olfactory explants.

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GABAA receptor stimulation promotes survival of embryonic rat striatal neurons in culture.

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Development of the GABA system in organotypic culture of hippocampal and cerebellar slices from a 67-kDa isoform of glutamic acid decarboxylase (GAD67)-deficient mice.

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Synaptic activity and the construction of cortical circuits

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Intracellular calcium increase induced by GABA in visual cortex of fetal and neonatal rats and its disappearance with development.

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GABA and glutamate depolarize cortical progenitor cells and inhibit DNA synthesis

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Transient expression of GABA immunoreactivity in the developing rat spinal cord

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Purification of vitamin D-dependent 28,000-Mr calcium-binding protein from bovine cerebellum and kidney by calcium-dependent elution from DEAE-cellulose DE-52 column chromatography.

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Excitatory and trophic action of GABA and related substances in newborn mice and organotypic cerebellar culture.

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Ornithine decarboxylase activity in fetal and newborn rat brain: responses to hypoxic and carbon monoxide hypoxia

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Neurotrophic effects of GABA in cultures of embryonic chick brain and retina

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Expression of gamma-aminobutyric acid and gonadotropin-releasing hormone during neuronal migration through the olfactory system.

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Identifiers

DOI

1 reference

9 February 2018

PubMed ID

10211762

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9 February 2018