Comparison of the effects of dopamine D1 and D2 receptor antagonists on nerve growth factor mRNA expression (Q48236274)

9 February 2018

title

Comparison of the effects of dopamine D1 and D2 receptor antagonists on nerve growth factor mRNA expression (English)

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9 February 2018

author name string

Ozaki T

series ordinal

1

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9 February 2018

Mui K

series ordinal

2

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9 February 2018

Yamagami S

series ordinal

3

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9 February 2018

language of work or name

English

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publication date

1 March 1999

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9 February 2018

European Journal of Pharmacology

published in

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9 February 2018

volume

369

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9 February 2018

issue

1

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9 February 2018

page(s)

133-143

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9 February 2018

Distorted distribution of nicotinamide-adenine dinucleotide phosphate-diaphorase neurons in temporal lobe of schizophrenics implies anomalous cortical development

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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The product of a fos-related gene, fra-1, binds cooperatively to the AP-1 site with Jun: transcription factor AP-1 is comprised of multiple protein complexes.

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Frontal cortical and left temporal glutamatergic dysfunction in schizophrenia

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Multiple signalling pathways interact in the regulation of nerve growth factor production in L929 fibroblasts

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D2 dopamine receptor antagonists induce fos and related proteins in rat striatal neurons

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c-Jun dimerizes with itself and with c-Fos, forming complexes of different DNA binding affinities

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Decreased hippocampal expression of a glutamate receptor gene in schizophrenia

7 January 2021

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Lesion-induced increase in nerve growth factor mRNA is mediated by c-fos

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Brain-derived neurotrophic factor is induced as an immediate early gene following N-methyl-D-aspartate receptor activation

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Paranoid schizophrenia may be caused by dopamine hyperactivity of CA1 hippocampus

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Clozapine and haloperidol produce a differential pattern of immediate early gene expression in rat caudate-putamen, nucleus accumbens, lateral septum and islands of Calleja

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Differential expression of inducible transcription factors in basal ganglia neurons.

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Theoretical Basis for a Pharmacology of Nerve Growth Factor Biosynthesis

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Regulation of nerve growth factor biosynthesis by beta-adrenergic receptor activation in astrocytoma cells: a potential role of c-Fos protein

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Stimulus-transcription coupling in the nervous system: involvement of the inducible proto-oncogenes fos and jun.

7 January 2021

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A gene activated by growth factors is related to the oncogene v-jun

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jun-D: a third member of the jun gene family

7 January 2021

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Mouse nerve growth factor gene: structure and expression

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Deficient NMDA-mediated glutamate release from synaptosomes of schizophrenics

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Different requirements for formation of Jun: Jun and Jun: Fos complexes

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Reduced [3H]flunitrazepam binding in cingulate cortex and hippocampus of postmortem schizophrenic brains: is selective loss of glutamatergic neurons associated with major psychoses?

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Glutamate receptor agonists increase the expression of Fos, Fra, and AP-1 DNA binding activity in the mammalian brain

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Implications of normal brain development for the pathogenesis of schizophrenia

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The product of a novel growth factor activated gene, fos B, interacts with JUN proteins enhancing their DNA binding activity

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Identification of two distinct regulatory regions adjacent to the human β-interferon gene

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10.1016/S0014-2999(99)00059-X

7 January 2021

Identifiers

DOI

1 reference

9 February 2018

1 reference

9 February 2018