Parallel dose-response studies of the voltage-dependent Na+ channel antagonist BW619C89, and the voltage-dependent Ca2+ channel antagonist nimodipine, in rat transient focal cerebral ischaemia (Q48290647)

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English	Parallel dose-response studies of the voltage-dependent Na+ channel antagonist BW619C89, and the voltage-dependent Ca2+ channel antagonist nimodipine, in rat transient focal cerebral ischaemia	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

Parallel dose-response studies of the voltage-dependent Na+ channel antagonist BW619C89, and the voltage-dependent Ca2+ channel antagonist nimodipine, in rat transient focal cerebral ischaemia (English)

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

cerebral infarction

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based on heuristic

inferred from title

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2

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

author name string

Kawaguchi K

1

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

Simon RP

3

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

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publication date

1 January 1999

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

European Journal of Pharmacology

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

364

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

2-3

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

99-105

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Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

Calcium channel blockers correct acidosis in ischemic rat brain without altering cerebral blood flow

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Effects of topical application of a calcium antagonist (nifedipine) on feline cortical pial microvasculature under normal conditions and in focal ischemia

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Correlation between amino acid release and neuropathologic outcome in rat brain following middle cerebral artery occlusion

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A comparison of the effects of a sodium channel blocker and an NMDA antagonist upon extracellular glutamate in rat focal cerebral ischemia

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Reperfusion following focal stroke hastens inflammation and resolution of ischemic injured tissue

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Nimodipine posttreatment does not increase blood flow in rats with focal cortical ischemia

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A controlled trial of nimodipine in acute ischemic stroke

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Continuous nimodipine treatment attenuates cortical infarction in rats subjected to 24 hours of focal cerebral ischemia.

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Induction of heat-shock protein (HSP72) in the cingulate and retrosplenial cortex by drugs that antagonize the effects of excitatory amino acids

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BW619C89, a glutamate release inhibitor, protects against focal cerebral ischemic damage

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Reversible middle cerebral artery occlusion without craniectomy in rats

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Placebo-controlled trial of nimodipine in the treatment of acute ischemic cerebral infarction

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Effect of pretreatment with the calcium antagonist nimodipine on local cerebral blood flow and histopathology after middle cerebral artery occlusion

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A randomized, double-blind, placebo-controlled ascending dose tolerance study of 619C89 in acute stroke

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NMDA Antagonist Neurotoxicity: Mechanism and Prevention

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Relationship between extracellular glutamate concentration and voltage-sensitive calcium channel function in focal cerebral ischemia in the rat.

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The glutamate antagonist MK-801 reduces focal ischemic brain damage in the rat

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Calcium fluxes, calcium antagonists, and calcium-related pathology in brain ischemia, hypoglycemia, and spreading depression: a unifying hypothesis

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Cerebroprotective effect of lamotrigine after focal ischemia in rats

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Long-term beneficial effects of BW619C89 on neurological deficit, cognitive deficit and brain damage after middle cerebral artery occlusion in the rat.

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A use-dependent sodium channel antagonist, 619C89, in reduction of ischemic brain damage and glutamate release after acute subdural hematoma in the rat.

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Identifiers

10.1016/S0014-2999(98)00827-9

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9932711

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