Dose-related effects of MK-801 on acute and chronic methylphenidate administration (Q48337908)

10 February 2018

title

Dose-related effects of MK-801 on acute and chronic methylphenidate administration (English)

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10 February 2018

author name string

Sripada S

1

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10 February 2018

Gaytan O

2

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10 February 2018

Al-rahim S

3

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10 February 2018

Swann A

4

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10 February 2018

Dafny N

5

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10 February 2018

language of work or name

English

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publication date

1 December 1998

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10 February 2018

number of pages

8

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based on heuristic

inferred from page(s)

published in

Brain Research

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10 February 2018

volume

814

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10 February 2018

issue

1-2

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10 February 2018

page(s)

78-85

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10 February 2018

Metabolic and experiential factors in the behavioral response to repeated amphetamine

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MK-801 disrupts the expression but not the development of bromocriptine sensitization: a state-dependency interpretation

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Multiple within-day conflict testing to define the time course of anxiolytic drug effects

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Time course effects of MK-801: the relationship between brain neurochemistry and behavior

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Trans-cranial electrical stimulation attenuates abrupt morphine withdrawal in rats assayed by remote computerized quantification of multiple motor behavior indices

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The development of behavioral sensitization to apomorphine is blocked by MK-801.

7 January 2021

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The topography of MK-801-induced locomotor patterns in rats

7 January 2021

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Sensitization to locomotor effects of methylphenidate in the rat

7 January 2021

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Comparison of the behavioral and biochemical effects of the NMDA receptor antagonists, MK-801 and phencyclidine

7 January 2021

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Inhibition of cocaine sensitization by MK-801, a noncompetitive N-methyl-D-aspartate (NMDA) receptor antagonist: evaluation by ambulatory activity in mice.

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MK-801 prevents the development of behavioral sensitization during repeated morphine administration

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Characteristics of the ambulation-increasing effect of the noncompetitive NMDA antagonist MK-801 in mice: assessment by the coadministration with central-acting drugs

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Effect of the NMDA receptor antagonist, MK-801, on locomotor activity and on the metabolism of dopamine in various brain areas of mice.

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A comparison of the motor-activating effects of acute and chronic exposure to amphetamine and methylphenidate

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Effects of MK-801 on spontaneous and amphetamine-stimulated dopamine release in striatum measured with in vivo microdialysis in awake rats

7 January 2021

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The effects of MK-801 on body temperature and behavior in the rat: cross-sensitization and cross-tolerance with phencyclidine

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L-NAME and MK-801 attenuate sensitization to the locomotor-stimulating effect of cocaine.

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Development of both conditioning and sensitization of the behavioral activating effects of amphetamine is blocked by the non-competitive NMDA receptor antagonist, MK-801.

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Competitive and non-competitive N-methyl-D-aspartate antagonists fail to prevent the induction of methamphetamine-induced sensitization

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MK-801 (dizocilpine): synergist and conditioned stimulus in bromocriptine-induced psychomotor sensitization.

7 January 2021

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Phencyclidine-like discriminative stimulus properties of MK-801 in rats

7 January 2021

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7 January 2021

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Coadministration of MK-801 with amphetamine, cocaine or morphine prevents rather than transiently masks the development of behavioral sensitization

7 January 2021

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Repeated administration of MK-801 produces sensitization to its own locomotor stimulant effects but blocks sensitization to amphetamine.

7 January 2021

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MK-801 does not prevent acute stimulatory effects of amphetamine or cocaine on locomotor activity or extracellular dopamine levels in rat nucleus accumbens.

7 January 2021

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NMDA antagonists make learning and recall state-dependent.

7 January 2021

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inferred from DOI database lookup

7 January 2021

based on heuristic

Does dizocilpine (MK-801) selectively block the enhanced responsiveness to repeated amphetamine administration?

1 reference

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inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-8993(98)01035-X

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10 February 2018

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10 February 2018