Cryopreservation of rat cortical synaptosomes and analysis of glucose and glutamate transporter activities, and mitochondrial function (Q48378727)

11 February 2018

title

Cryopreservation of rat cortical synaptosomes and analysis of glucose and glutamate transporter activities, and mitochondrial function (English)

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11 February 2018

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2

D A Butterfield

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J G Begley

1

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J N Keller

3

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T Koppal

4

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J Drake

5

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M P Mattson

6

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publication date

1 September 1998

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11 February 2018

published in

Brain Research Protocols

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volume

3

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issue

1

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page(s)

76-82

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beta-Amyloid-associated free radical oxidative stress and neurotoxicity: implications for Alzheimer's disease

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Cryopreservation of freshly isolated synaptosomes prepared from the cerebral cortex of rats

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Functional interactions between neurons and astrocytes I. Turnover and metabolism of putative amino acid transmitters

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Diminished synaptosomal dopamine (DA) release and DA autoreceptor supersensitivity in schizophrenia

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Prevention of hyperoxia-induced alterations in synaptosomal membrane-associated proteins by N-tert-butyl-alpha-phenylnitrone and 4-hydroxy-2,2,6,6-tetramethylpiperidin-1-oxyl (Tempol).

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4-Hydroxynonenal, an aldehydic product of membrane lipid peroxidation, impairs glutamate transport and mitochondrial function in synaptosomes

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Impairment of glucose and glutamate transport and induction of mitochondrial oxidative stress and dysfunction in synaptosomes by amyloid beta-peptide: role of the lipid peroxidation product 4-hydroxynonenal

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Manifestations of cell damage after freezing and thawing.

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Inhibition of [3H]dopamine uptake into striatal synaptosomes by isoquinoline derivatives structurally related to 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

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Release of glutamate, aspartate, and gamma-aminobutyric acid from isolated nerve terminals

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Nitric oxide inhibits 3H-glutamate transport in synaptosomes.

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The intracellular component of cellular 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) reduction is specifically inhibited by beta-amyloid peptides.

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Deficient NMDA-mediated glutamate release from synaptosomes of schizophrenics

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Mitochondrial depolarization in glutamate-stimulated neurons: an early signal specific to excitotoxin exposure

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7 January 2021

Thirty years of synaptosome research

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7 January 2021

10.1016/S1385-299X(98)00024-5

Identifiers

DOI

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11 February 2018

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11 February 2018