Fas has a crucial role in the progression of experimental autoimmune encephalomyelitis. (Q48390583)

11 February 2018

title

Fas has a crucial role in the progression of experimental autoimmune encephalomyelitis. (English)

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11 February 2018

author name string

Okuda Y

1

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11 February 2018

Bernard CC

2

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11 February 2018

Fujimura H

3

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11 February 2018

Yanagihara T

4

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11 February 2018

Sakoda S

5

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11 February 2018

publication date

1 April 1998

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11 February 2018

published in

Molecular Immunology

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11 February 2018

volume

35

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11 February 2018

issue

5

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11 February 2018

page(s)

317-326

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11 February 2018

Aberrant transcription caused by the insertion of an early transposable element in an intron of the Fas antigen gene of lpr mice

cites work

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7 January 2021

Expression, purification, and encephalitogenicity of recombinant human myelin oligodendrocyte glycoprotein.

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Multiple sclerosis: oligodendrocytes display cell death-related molecules in situ but do not undergo apoptosis.

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Regulatory T cell clones induced by oral tolerance: suppression of autoimmune encephalomyelitis

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7 January 2021

The role of Fas in autoimmune diabetes

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7 January 2021

Involvement of the CD95 (APO-1/Fas) receptor/ligand system in multiple sclerosis brain

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7 January 2021

Multiple sclerosis: Fas signaling in oligodendrocyte cell death

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7 January 2021

Increased Fas antigen on T cells in multiple sclerosis

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7 January 2021

Antibody response in Lewis rats injected with myelin oligodendrocyte glycoprotein derived peptides

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7 January 2021

Detection of the soluble form of the Fas molecule in patients with multiple sclerosis and human T-lymphotropic virus type I-associated myelopathy

1 reference

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7 January 2021

Requirement of Fas for the development of autoimmune diabetes in nonobese diabetic mice

1 reference

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7 January 2021

Oral tolerance to myelin basic protein and natural recovery from experimental autoimmune encephalomyelitis are associated with downregulation of inflammatory cytokines and differential upregulation of transforming growth factor beta, interleukin 4,

1 reference

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7 January 2021

T cells specific for the myelin oligodendrocyte glycoprotein mediate an unusual autoimmune inflammatory response in the central nervous system.

1 reference

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7 January 2021

A myelin oligodendrocyte glycoprotein peptide induces typical chronic experimental autoimmune encephalomyelitis in H-2b mice: fine specificity and T cell receptor V beta expression of encephalitogenic T cells

1 reference

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7 January 2021

Fas and Fas ligand: a death factor and its receptor.

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7 January 2021

Expression of the inducible isoform of nitric oxide synthase in the central nervous system of mice correlates with the severity of actively induced experimental allergic encephalomyelitis

1 reference

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7 January 2021

Nitric oxide via an inducible isoform of nitric oxide synthase is a possible factor to eliminate inflammatory cells from the central nervous system of mice with experimental allergic encephalomyelitis

1 reference

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7 January 2021

The development of autoimmune encephalomyelitis provoked by myelin oligodendrocyte glycoprotein is associated with an upregulation of both proinflammatory and immunoregulatory cytokines in the central nervous system.

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7 January 2021

The Dale E. McFarlin Memorial Lecture: the immunology of the multiple sclerosis lesion

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7 January 2021

Autoimmune gld mutation uncouples suicide and cytokine/proliferation pathways in activated, mature T cells

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7 January 2021

Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide

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7 January 2021

Apoptotic elimination of V beta 8.2+ cells from the central nervous system during recovery from experimental autoimmune encephalomyelitis induced by the passive transfer of V beta 8.2+ encephalitogenic T cells

1 reference

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7 January 2021

Antigen-specific down-regulation of myelin basic protein-reactive T cells during spontaneous recovery from experimental autoimmune encephalomyelitis: further evidence of apoptotic deletion of autoreactive T cells in the central nervous system

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7 January 2021

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

1 reference

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7 January 2021

Two models for multiple sclerosis: experimental allergic encephalomyelitis and Theiler's murine encephalomyelitis virus

1 reference

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7 January 2021

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor

1 reference

https://api.crossref.org/works/10.1016%2FS0161-5890%2898%2900049-2

7 January 2021

10.1016/S0161-5890(98)00049-2

Identifiers

DOI

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11 February 2018

PubMed ID

9747891

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11 February 2018