Mice expressing human mutant presenilin-1 exhibit decreased activation of NF-kappaB p50 in hippocampal neurons after injury (Q48393631)

11 February 2018

title

Mice expressing human mutant presenilin-1 exhibit decreased activation of NF-kappaB p50 in hippocampal neurons after injury (English)

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11 February 2018

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Kassed CA

1

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11 February 2018

Butler TL

2

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11 February 2018

Navidomskis MT

3

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11 February 2018

Gordon MN

4

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11 February 2018

Morgan D

5

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Pennypacker KR

6

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11 February 2018

publication date

1 January 2003

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11 February 2018

published in

Molecular Brain Research

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11 February 2018

volume

110

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issue

1

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page(s)

152-157

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11 February 2018

A 65-kappaD subunit of active NF-kappaB is required for inhibition of NF-kappaB by I kappaB.

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Functional phenotype in transgenic mice expressing mutant human presenilin-1.

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Role of nuclear factor kappa B in neuropathological mechanisms

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Increased amyloid-beta42(43) in brains of mice expressing mutant presenilin 1

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Reversible activation of nuclear factor-kappaB in human end-stage heart failure after left ventricular mechanical support

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Secreted beta-amyloid precursor protein counteracts the proapoptotic action of mutant presenilin-1 by activation of NF-kappaB and stabilization of calcium homeostasis

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Iodoacetate protects hippocampal neurons against excitotoxic and oxidative injury: involvement of heat-shock proteins and Bcl-2.

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Intramolecular masking of the nuclear location signal and dimerization domain in the precursor for the p50 NF-kappa B subunit

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7 January 2021

Presenilin 1 suppresses the function of c-Jun homodimers via interaction with QM/Jif-1

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Brain synapses contain inducible forms of the transcription factor NF-kappa B.

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Constitutive NF-kappa B activity in neurons

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Lack of NF-kappaB p50 exacerbates degeneration of hippocampal neurons after chemical exposure and impairs learning

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Negative regulation of the SAPK/JNK signaling pathway by presenilin 1

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NF-kappaB as a therapeutic target in chronic inflammation: recent advances

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Amyloid precursor proteins protect neurons of transgenic mice against acute and chronic excitotoxic injuries in vivo.

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Roles of nuclear factor kappaB in neuronal survival and plasticity

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Presenilin 1 regulates pharmacologically distinct gamma -secretase activities. Implications for the role of presenilin in gamma -secretase cleavage

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NF-kappaB p50 is increased in neurons surviving hippocampal injury

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Fluoro-Jade: novel fluorochromes for detecting toxicant-induced neuronal degeneration

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Fluoro-Jade: a novel fluorochrome for the sensitive and reliable histochemical localization of neuronal degeneration

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TNF activates NF-kappa B by phosphatidylcholine-specific phospholipase C-induced "acidic" sphingomyelin breakdown

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In search of gamma-secretase: presenilin at the cutting edge

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Generation of amyloid beta protein from a presenilin-1 and betaAPP complex

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Secreted forms of beta-amyloid precursor protein protect against ischemic brain injury

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Participation of presenilin 2 in apoptosis: enhanced basal activity conferred by an Alzheimer mutation

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Nuclear uptake control of NF-kappa B by MAD-3, an I kappa B protein present in the nucleus

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7 January 2021

10.1016/S0169-328X(02)00658-7

Identifiers

DOI

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11 February 2018

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11 February 2018