Role of high mobility group protein-1 (HMG1) in amyloid-beta homeostasis. (Q48396116)

11 February 2018

title

Role of high mobility group protein-1 (HMG1) in amyloid-beta homeostasis. (English)

1 reference

11 February 2018

author

George Perry

8

1 reference

11 February 2018

Mark Anthony Smith

7

Mark A Smith

1 reference

11 February 2018

9

Shun Shimohama

1 reference

11 February 2018

Yoshihisa Kitamura

2

Yoshihisa Kitamura

1 reference

11 February 2018

Daiju Tsuchiya

5

Daiju Tsuchiya

1 reference

11 February 2018

Kazuyuki Takata

1

Kazuyuki Takata

1 reference

11 February 2018

Keiichi Shibagaki

4

Keiichi Shibagaki

1 reference

11 February 2018

author name string

Jun-ichi Kakimura

3

1 reference

11 February 2018

Takashi Taniguchi

6

1 reference

11 February 2018

language of work or name

English

0 references

publication date

1 February 2003

1 reference

11 February 2018

Biochemical and Biophysical Research Communications

published in

1 reference

11 February 2018

volume

301

1 reference

11 February 2018

issue

3

1 reference

11 February 2018

page(s)

699-703

1 reference

11 February 2018

Translating cell biology into therapeutic advances in Alzheimer's disease

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Amyloid-beta immunotherapy for Alzheimer's disease: the end of the beginning

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Peripherally administered antibodies against amyloid beta-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

TGF-beta1 promotes microglial amyloid-beta clearance and reduces plaque burden in transgenic mice.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Bip/GRP78-induced production of cytokines and uptake of amyloid-beta(1-42) peptide in microglia

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Microglial activation and amyloid-beta clearance induced by exogenous heat-shock proteins.

7 January 2021

2 references

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

7 January 2021

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

inferred from DOI database lookup

7 January 2021

based on heuristic

Structural features of the HMG chromosomal proteins and their genes

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

The lack of chromosomal protein Hmg1 does not disrupt cell growth but causes lethal hypoglycaemia in newborn mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

HMG-1 as a late mediator of endotoxin lethality in mice

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

The receptor for advanced glycation end products (RAGE) is a cellular binding site for amphoterin. Mediation of neurite outgrowth and co-expression of rage and amphoterin in the developing nervous system

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Alteration of proteins regulating apoptosis, Bcl-2, Bcl-x, Bax, Bak, Bad, ICH-1 and CPP32, in Alzheimer's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Amphoterin includes a sequence motif which is homologous to the Alzheimer's beta-amyloid peptide (Abeta), forms amyloid fibrils in vitro, and binds avidly to Abeta.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-291X%2803%2900024-X

10.1016/S0006-291X(03)00024-X

7 January 2021

Identifiers

DOI

1 reference

11 February 2018

PubMed ID

12565837

1 reference

11 February 2018