Predictive and Prognostic Roles of Abnormal Expression of Tissue miR-125b, miR-221, and miR-222 in Glioma. (Q48414232)

11 February 2018

title

Predictive and Prognostic Roles of Abnormal Expression of Tissue miR-125b, miR-221, and miR-222 in Glioma. (English)

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11 February 2018

author name string

Xinxing Li

1

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11 February 2018

Jihui Zheng

2

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11 February 2018

Liangyu Chen

3

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11 February 2018

Hongyu Diao

4

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11 February 2018

Yunhui Liu

5

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11 February 2018

publication date

11 December 2014

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11 February 2018

published in

Molecular Neurobiology

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11 February 2018

volume

53

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11 February 2018

issue

1

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11 February 2018

page(s)

577-583

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11 February 2018

https://scigraph.springernature.com/pub.10.1007/s12035-014-9017-x

exact match

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cites work

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miR-125b regulates the proliferation of glioblastoma stem cells by targeting E2F2.

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MiR-221 and miR-222 target PUMA to induce cell survival in glioblastoma

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miR-125b inhibits Connexin43 and promotes glioma growth

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Myc-associated zinc finger protein (MAZ) is regulated by miR-125b and mediates VEGF-induced angiogenesis in glioblastoma.

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MicroRNAs 221 and 222 bypass quiescence and compromise cell survival

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miR-221/222 overexpession in human glioblastoma increases invasiveness by targeting the protein phosphate PTPμ

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miR-221/222 is the regulator of Cx43 expression in human glioblastoma cells

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MicroRNA-222 promotes tumorigenesis via targeting DKK2 and activating the Wnt/β-catenin signaling pathway

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Downregulation of miR-221/222 sensitizes glioma cells to temozolomide by regulating apoptosis independently of p53 status.

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miR-221/222 confers radioresistance in glioblastoma cells through activating Akt independent of PTEN status

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Co-suppression of miR-221/222 cluster suppresses human glioma cell growth by targeting p27kip1 in vitro and in vivo

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[miR-221 mediates epithelial-mesenchymal transition-related gene expressions via regulation of PTEN/Akt signaling in drug-resistant glioma cells].

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Identifiers

DOI

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11 February 2018

PubMed ID

25502291

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11 February 2018