Astrocyte-induced T cell elimination is CD95 ligand dependent. (Q48453594)

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14 December 2019

title

Astrocyte-induced T cell elimination is CD95 ligand dependent (English)

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14 December 2019

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3

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14 December 2019

Susanne Wolf

5

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14 December 2019

Frauke Zipp

8

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14 December 2019

Barbara Steiner

2

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14 December 2019

author name string

Ingo Bechmann

1

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14 December 2019

Gil Mor

4

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Martin Beyer

6

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14 December 2019

Robert Nitsch

7

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14 December 2019

publication date

1 November 2002

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Journal of Neuroimmunology

14 December 2019

published in

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volume

132

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14 December 2019

issue

1-2

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page(s)

60-65

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T-cell apoptosis in inflammatory brain lesions: destruction of T cells does not depend on antigen recognition

14 December 2019

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FasL (CD95L, Apo1L) is expressed in the normal rat and human brain: evidence for the existence of an immunological brain barrier.

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Reactive astrocytes upregulate Fas (CD95) and Fas ligand (CD95L) expression but do not undergo programmed cell death during the course of anterograde degeneration

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Antigen presentation by astrocytes primes rat T lymphocytes for apoptotic cell death. A model for T-cell apoptosis in vivo.

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Macrophage recruitment to acutely injured central nervous system is inhibited by a resident factor: a basis for an immune-brain barrier

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Accumulation of passively transferred primed T cells independently of their antigen specificity following central nervous system trauma.

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Coexpression of Fas/FasL and Bax on brain and infiltrating T cells in the central nervous system is closely associated with apoptotic cell death during autoimmune encephalomyelitis

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Interaction between apoptotic cells and reactive brain cells in the central nervous system of rats with autoimmune encephalomyelitis.

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Fas ligand expression by astrocytoma in vivo: maintaining immune privilege in the brain?

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Dual role for Fas ligand in the initiation of and recovery from experimental allergic encephalomyelitis

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Fas-mediated apoptosis in clinical remissions of relapsing experimental autoimmune encephalomyelitis

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7 January 2021

Apoptotic elimination of V beta 8.2+ cells from the central nervous system during recovery from experimental autoimmune encephalomyelitis induced by the passive transfer of V beta 8.2+ encephalitogenic T cells

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7 January 2021

The roles of Fas, Fas ligand and Bcl-2 in T cell apoptosis in the central nervous system in experimental autoimmune encephalomyelitis

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A targeted DNA vaccine encoding fas ligand defines its dual role in the regulation of experimental autoimmune encephalomyelitis

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Identifiers

DOI

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14 December 2019

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