TrkA receptors delay C6-2B glioma cell growth in rat striatum. (Q48462004)

11 February 2018

title

TrkA receptors delay C6-2B glioma cell growth in rat striatum. (English)

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11 February 2018

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Rabin SJ

1

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11 February 2018

Tornatore C

2

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11 February 2018

Baker-Cairns B

3

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11 February 2018

Spiga G

4

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11 February 2018

Mocchetti I

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11 February 2018

publication date

1 May 1998

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11 February 2018

published in

Molecular Brain Research

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11 February 2018

volume

56

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11 February 2018

issue

1-2

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11 February 2018

page(s)

273-276

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11 February 2018

Patient age, histologic features, and length of survival in patients with glioblastoma multiforme.

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TrkA cross-linking mimics neuronal responses to nerve growth factor

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Induction of nerve growth factor responsiveness in C6-2B glioma cells by expression of trkA proto-oncogene

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The trk Proto-Oncogene Product: a Signal Transducing Receptor for Nerve Growth Factor

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The nerve growth factor 35 years later

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Expression of trkA cDNA in neuroblastomas mediates differentiation in vitro and in vivo

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Nerve growth factor suppresses the transforming phenotype of human prolactinomas

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Regulation of nerve growth factor biosynthesis by beta-adrenergic receptor activation in astrocytoma cells: a potential role of c-Fos protein

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Glucocorticoids differentially increase nerve growth factor and basic fibroblast growth factor expression in the rat brain.

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Nerve growth factor induces apoptosis in human medulloblastoma cell lines that express TrkA receptors

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Genetically modified skin fibroblasts persist long after transplantation but gradually inactivate introduced genes

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Advances in radiation therapy for brain tumors

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Engraftment of C6-2B cells into the striatum of ACI nude rats as a tool for comparison of the in vitro and in vivo phenotype of a glioma cell line

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Identifiers

DOI

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11 February 2018

PubMed ID

9602149

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11 February 2018