Cell death and associated c-jun induction in perinatal hypoxia-ischemia. Effect of the neuroprotective drug dexamethasone (Q48462416)

11 February 2018

title

Cell death and associated c-jun induction in perinatal hypoxia-ischemia. Effect of the neuroprotective drug dexamethasone (English)

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11 February 2018

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A Macaya

1

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11 February 2018

F Munell

2

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11 February 2018

I Ferrer

3

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11 February 2018

C de Torres

4

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11 February 2018

J Reventós

5

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11 February 2018

publication date

1 May 1998

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11 February 2018

published in

Molecular Brain Research

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11 February 2018

volume

56

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11 February 2018

issue

1-2

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11 February 2018

page(s)

29-37

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11 February 2018

Oncogene jun encodes a sequence-specific trans-activator similar to AP-1.

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Dexamethasone Prevents Hypoxic-Ischemic Brain Damage in the Neonatal Rat

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Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal loss

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Regional expression of c-fos and heat shock protein-70 mRNA following hypoxia-ischemia in immature rat brain

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A comparison of the protective effect of dexamethasone to other potential prophylactic agents in a neonatal rat model of cerebral hypoxia-ischemia

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Axotomized medial septal-diagonal band neurons express Jun-like immunoreactivity

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The role of inducible transcription factors in apoptotic nerve cell death

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Immediate-early gene protein expression in neurons undergoing delayed death, but not necrosis, following hypoxic-ischaemic injury to the young rat brain

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Dexamethasone prevents apoptosis in a neonatal rat model of hypoxic-ischemic encephalopathy (HIE) by a reactive oxygen species-independent mechanism.

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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The differential control of c-jun expression in regenerating sensory neurons and their associated glial cells

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Naturally occurring cell death in the developing cerebral cortex of the rat. Evidence of apoptosis-associated internucleosomal DNA fragmentation

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7 January 2021

Evidence of nuclear DNA fragmentation following hypoxia-ischemia in the infant rat brain, and transient forebrain ischemia in the adult gerbil

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Selective c-Jun overexpression is associated with ionizing radiation-induced apoptosis in the developing cerebellum of the rat

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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DNA fragmentation and activation of c-Jun in the cerebellum of mutant mice (weaver, Purkinje cell degeneration).

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Transient c-fos expression accompanies naturally occurring cell death in the developing interhemispheric cortex of the rat

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Immediate early gene induction after neonatal hypoxia-ischemia

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The Effects of Corticosteroids on Myelination of the Developing Rat Brain

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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The transcription factors c-JUN, JUN D and CREB, but not FOS and KROX-24, are differentially regulated in axotomized neurons following transection of rat sciatic nerve

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7 January 2021

DNA fragmentation indicative of apoptosis following unilateral cerebral hypoxia-ischemia in the neonatal rat

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Viability thresholds and the penumbra of focal ischemia

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Brain-derived neurotrophic factor is induced as an immediate early gene following N-methyl-D-aspartate receptor activation

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Induction of fos-like immunoreactivity in hypothalamic corticotropin-releasing factor neurons after adrenalectomy in the rat.

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Long-term increase in the levels of c-jun mRNA and jun protein-like immunoreactivity in motor and sensory neurons following axon damage

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Kainic acid-induced neuronal death is associated with DNA damage and a unique immediate-early gene response in c-fos-lacZ transgenic rats

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Adrenalectomy potentiates immediate early gene expression in rat brain

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7 January 2021

Apoptotic DNA fragmentation in the rat cerebral cortex induced by permanent middle cerebral artery occlusion

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7 January 2021

Mutual transrepression of Fos and the glucocorticoid receptor: involvement of a functional domain in Fos which is absent in FosB

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7 January 2021

Apoptosis in substantia nigra following developmental striatal excitotoxic injury

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7 January 2021

Adrenal steroid receptors and actions in the nervous system

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7 January 2021

Localization of c-fos, c-jun, and hsp70 mRNA expression in brain after neonatal hypoxia-ischemia

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7 January 2021

Delayed neuronal death in the CA1 pyramidal cell layer of the gerbil hippocampus following transient ischemia is apoptosis.

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7 January 2021

Apoptosis in substantia nigra following developmental hypoxic-ischemic injury.

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7 January 2021

The influence of immaturity on hypoxic‐ischemic brain damage in the rat

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7 January 2021

The role of Jun transcription factor expression and phosphorylation in neuronal differentiation, neuronal cell death, and plastic adaptations in vivo

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7 January 2021

Functional antagonism between oncoprotein c-Jun and the glucocorticoid receptor

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7 January 2021

Continuous c-fos expression precedes programmed cell death in vivo

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7 January 2021

Apoptosis and steroid hormones

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7 January 2021

Identification of necrotic cell death by the TUNEL assay in the hypoxic-ischemic neonatal rat brain

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7 January 2021

Glucocorticoid prevention of neonatal hypoxic-ischemic damage: role of hyperglycemia and antioxidant enzymes

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7 January 2021

Dexamethasone prevents cerebral infarction without affecting cerebral blood flow in neonatal rats

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7 January 2021

Prevention of hypoxic-ischemic damage with dexamethasone is dependent on age and not influenced by fasting.

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7 January 2021

Brain damage induced by prenatal exposure to dexamethasone in fetal rhesus macaques. I. Hippocampus

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7 January 2021

Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation

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7 January 2021

Transcriptional interference between c-Jun and the glucocorticoid receptor: mutual inhibition of DNA binding due to direct protein-protein interaction.

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7 January 2021

Apoptotic Cells in the Human Endometrium and Placental Villi: Pitfalls in Applying the TUNEL Method

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7 January 2021

10.1016/S0169-328X(98)00024-2

Identifiers

DOI

1 reference

11 February 2018

1 reference

11 February 2018