Pathological relationships between microglial cell activity and tau and amyloid beta protein in patients with Alzheimer's disease. (Q48466773)

11 February 2018

title

Pathological relationships between microglial cell activity and tau and amyloid beta protein in patients with Alzheimer's disease. (English)

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11 February 2018

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Stuart Pickering-Brown

4

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11 February 2018

author name string

Hayes A

1

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11 February 2018

Thaker U

2

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11 February 2018

Iwatsubo T

3

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11 February 2018

Mann DM

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11 February 2018

language of work or name

English

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publication date

1 October 2002

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11 February 2018

published in

Neuroscience Letters

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11 February 2018

volume

331

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11 February 2018

issue

3

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11 February 2018

page(s)

171-174

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11 February 2018

Microglia, amyloid and dementia in alzheimer disease. A correlative study.

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Brain interleukin 1 and S-100 immunoreactivity are elevated in Down syndrome and Alzheimer disease

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Microglial interleukin-1 alpha expression in human head injury: correlations with neuronal and neuritic beta-amyloid precursor protein expression

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Inflammatory mechanisms in Alzheimer's disease

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Relationship of microglia and astrocytes to amyloid deposits of Alzheimer disease.

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Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43).

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Microglial cells and amyloid beta protein (A beta) deposition; association with A beta 40-containing plaques

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Preferential deposition of amyloid beta protein (Abeta) in the form Abeta40 in Alzheimer's disease is associated with a gene dosage effect of the apolipoprotein E E4 allele.

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The Consortium to Establish a Registry for Alzheimer's Disease (CERAD). Part II. Standardization of the neuropathologic assessment of Alzheimer's disease

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Interleukin-1 promotion of MAPK-p38 overexpression in experimental animals and in Alzheimer's disease: potential significance for tau protein phosphorylation

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Progression of neurofibrillary changes and PHF-tau in end-stage Alzheimer's disease is different from plaque and cortical microglial pathology

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Potential mechanisms of interleukin-1 involvement in cerebral ischaemia.

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The complex of microglial cells and amyloid star in three-dimensional reconstruction.

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Plaques, tangles and dementia. A quantitative study.

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Identifiers

DOI

1 reference

11 February 2018

release_v5uopxyktbdyxklt6p2j7tiigu

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/v5uopxyktbdyxklt6p2j7tiigu

24 November 2022

mapped directly with Wikidata item

PubMed ID

12383924

1 reference

11 February 2018