Involvement of protease-activated receptor-1 in the in vitro development of mesencephalic dopaminergic neurons. (Q48522972)

title

Involvement of protease-activated receptor-1 in the in vitro development of mesencephalic dopaminergic neurons. (English)

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12 February 2018

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T Debeir

1

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12 February 2018

J Benavides

series ordinal

2

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12 February 2018

X Vigé

series ordinal

3

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12 February 2018

language of work or name

English

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publication date

1 February 1998

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12 February 2018

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12 February 2018

volume

82

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12 February 2018

page(s)

739-752

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12 February 2018

issue

3

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12 February 2018

Thrombin receptor peptides induce shape change in neonatal murine astrocytes in culture.

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Transduction mechanisms involved in thrombin receptor-induced nerve growth factor secretion and cell division in primary cultures of astrocytes

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Basic fibroblast growth factor promotes the survival and development of mesencephalic neurons in culture

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Thrombin, its receptor and protease nexin I, its potent serpin, in the nervous system

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Thrombin modulates and reverses neuroblastoma neurite outgrowth

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BDNF is a neurotrophic factor for dopaminergic neurons of the substantia nigra.

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Overlapping and distinct actions of the neurotrophins BDNF, NT-3, and NT-4/5 on cultured dopaminergic and GABAergic neurons of the ventral mesencephalon.

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Thrombin receptor activation causes rapid neural cell rounding and neurite retraction independent of classic second messengers

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Promotion of central cholinergic and dopaminergic neuron differentiation by brain-derived neurotrophic factor but not neurotrophin 3.

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Proteolytic activity, synapse elimination, and the Hebb synapse

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Enhancement of the synthesis and secretion of nerve growth factor in primary cultures of glial cells by proteases: a possible involvement of thrombin.

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cDNA cloning and expression of a hamster alpha-thrombin receptor coupled to Ca2+ mobilization.

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Thrombin receptor antagonists

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Sequential effects of astroglial-derived factors on neurite outgrowth: initiation by protease inhibitors and potentiation by extracellular matrix components

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Opposing actions of thrombin and protease nexin-1 on amyloid beta-peptide toxicity and on accumulation of peroxides and calcium in hippocampal neurons.

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Thrombin causes neurite retraction in neuronal cells through activation of cell surface receptors

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Soluble striatal extracts enhance development of mesencephalic dopaminergic neurons in vitro.

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Thrombin receptor activation protects neurons and astrocytes from cell death produced by environmental insults.

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Molecular cloning of a functional thrombin receptor reveals a novel proteolytic mechanism of receptor activation

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Protease nexin-1, an antithrombin with neurite outgrowth activity, is reduced in Alzheimer disease

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Cellular localization of thrombin receptor mRNA in rat brain: expression by mesencephalic dopaminergic neurons and codistribution with prothrombin mRNA.

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7 January 2021

10.1016/S0306-4522(97)00317-5

Identifiers

DOI

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12 February 2018

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12 February 2018