Striatal and urinary DOPAC/DA ratio may indicate a long-lasting DA release enhancement by MPP+ and MPTP. (Q48527328)

12 February 2018

title

Striatal and urinary DOPAC/DA ratio may indicate a long-lasting DA release enhancement by MPP+ and MPTP. (English)

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12 February 2018

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S P Bagchi

1

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12 February 2018

publication date

1 February 1998

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12 February 2018

published in

Neurochemical Research

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12 February 2018

volume

23

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12 February 2018

issue

2

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12 February 2018

page(s)

127-134

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12 February 2018

Effect of selective destruction of central and peripheral catecholamine-containing neurones with 6-hydroxydopamine on catecholamine excretion in the rat.

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MPP+-induced efflux of dopamine and lactate from rat striatum have similar time courses as shown by in vivo brain dialysis

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Potentiation by reserpine and tetrabenazine of brain catecholamine depletions by MPTP (1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine) in the mouse; evidence for subcellular sequestration as basis for cellular resistance to the toxicant

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Trace dosages of the neurotoxins MPTP and MPP+ may affect brain dopamine in vivo

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Selective MPP+ uptake into synaptic dopamine vesicles: possible involvement in MPTP neurotoxicity

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Peripheral distribution of free dopamine and its metabolites in the rat

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Advances in our understanding of the mechanisms of the neurotoxicity of MPTP and related compounds

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Tissue concentrations of MPTP and MPP+ in relation to catecholamine depletion after the oral or subcutaneous administration of MPTP to mice

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Brain dialysis in conscious rats reveals an instantaneous massive release of striatal dopamine in response to MPP+.

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Selective nigral toxicity after systemic administration of 1-methyl-4-phenyl-1,2,5,6-tetrahydropyrine (MPTP) in the squirrel monkey

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Evidence for an increased catecholamine synthesis in rat adrenal glands following stimulation of peripheral dopamine receptors

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Amphetamine, but not reserpine, protects mice against dopaminergic neurotoxicity of MPTP.

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12 December 2020

Intraneuronal generation of a pyridinium metabolite may cause drug-induced parkinsonism

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12 December 2020

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12 December 2020

Reserpine induced intraneuronal dopamine oxidation: reversal by MPP+ action

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12 December 2020

A primate model of parkinsonism: selective destruction of dopaminergic neurons in the pars compacta of the substantia nigra by N-methyl-4-phenyl-1,2,3,6-tetrahydropyridine

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In vivo measurement of extracellular dopamine and DOPAC in rat striatum after various dopamine-releasing drugs; implications for the origin of extracellular DOPAC.

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12 December 2020

Effects of MPP+ on catecholamine levels in adrenal glands and heart of rats

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12 December 2020

Correlation between 1-methyl-4-phenylpyridinium ion (MPP+) levels, ascorbic acid oxidation and glutathione levels in the striatal synaptosomes of the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP)-treated rat

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12 December 2020

1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine-induced parkinsonism in the common marmoset

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12 December 2020

The effects of 1-methyl-4-phenylpyridinium ion (MPP+) on the efflux and metabolism of endogenous dopamine in rat striatal slices

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12 December 2020

Identifiers

DOI

10.1023/A:1022464421655

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12 February 2018

Dimensions Publication ID

1020111539

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12 February 2018