Inhibition of striatal energy metabolism produces cell loss in the ipsilateral substantia nigra. (Q48576196)

12 February 2018

title

Inhibition of striatal energy metabolism produces cell loss in the ipsilateral substantia nigra. (English)

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12 February 2018

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Sonsalla PK

1

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12 February 2018

Manzino L

2

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12 February 2018

Sinton CM

3

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12 February 2018

Liang CL

4

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12 February 2018

German DC

5

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12 February 2018

Zeevalk GD

6

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12 February 2018

language of work or name

English

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publication date

1 October 1997

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12 February 2018

number of pages

4

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based on heuristic

inferred from page(s)

published in

Brain Research

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12 February 2018

volume

773

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12 February 2018

issue

1-2

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12 February 2018

page(s)

223-226

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12 February 2018

Damage to dopaminergic nerve terminals in mice by combined treatment of intrastriatal malonate with systemic methamphetamine or MPTP

cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Electron transfer complexes I and IV of platelets are abnormal in Parkinson's disease but normal in Parkinson-plus syndromes.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Respiratory chain abnormalities in skeletal muscle from patients with Parkinson's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Age-dependent vulnerability of the striatum to the mitochondrial toxin 3-nitropropionic acid.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

The neurotoxin MPTP causes degeneration of specific nucleus A8, A9 and A10 dopaminergic neurons in the mouse

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Enzymatic oxidation of dopamine: the role of prostaglandin H synthase

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Platelet mitochondrial function in Parkinson's disease. The Royal Kings and Queens Parkinson Disease Research Group

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

The high sensitivity to rotenone of striatal dopamine uptake suggests the existence of a constitutive metabolic deficiency in dopaminergic neurons from the substantia nigra.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

A selective toxicity toward cultured mesencephalic dopaminergic neurons is induced by the synergistic effects of energetic metabolism impairment and NMDA receptor activation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

An immunohistochemical study on alpha-ketoglutarate dehydrogenase complex in Parkinson's disease.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Progressive degeneration of nigrostriatal dopamine neurons following intrastriatal terminal lesions with 6-hydroxydopamine: a combined retrograde tracing and immunocytochemical study in the rat.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Origin and functional consequences of the complex I defect in Parkinson's disease

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Oxygen radical mechanisms of brain injury following ischemia and reperfusion

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

In vivo vulnerability of dopamine neurons to inhibition of energy metabolism.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

10.1016/S0006-8993(97)00941-4

7 January 2021

Identifiers

DOI

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12 February 2018

PubMed ID

9409726

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12 February 2018