Accumulation of the inhibitory receptor EphA4 may prevent regeneration of corticospinal tract axons following lesion. (Q48579237)

13 February 2018

title

Accumulation of the inhibitory receptor EphA4 may prevent regeneration of corticospinal tract axons following lesion. (English)

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13 February 2018

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Jez Fabes

1

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13 February 2018

Patrick Anderson

2

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13 February 2018

Rafael J Yáñez-Muñoz

3

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13 February 2018

Adrian Thrasher

4

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13 February 2018

Caroline Brennan

5

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13 February 2018

Stephen Bolsover

6

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13 February 2018

language of work or name

English

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publication date

1 April 2006

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13 February 2018

European Journal of Neuroscience

published in

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13 February 2018

volume

23

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13 February 2018

issue

7

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13 February 2018

page(s)

1721-1730

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13 February 2018

EphB receptors and ephrin-B ligands regulate spinal sensory connectivity and modulate pain processing.

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Ephrin-B3 is a myelin-based inhibitor of neurite outgrowth

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Semiquantitative expression analysis of ephrine-receptor tyrosine kinase mRNA's in a rat model of traumatic brain injury.

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Chondroitinase ABC promotes functional recovery after spinal cord injury

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Recovery from spinal cord injury mediated by antibodies to neurite growth inhibitors

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Ephrins in reverse, park and drive

21 January 2018

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High-level transduction and gene expression in hematopoietic repopulating cells using a human immunodeficiency [correction of imunodeficiency] virus type 1-based lentiviral vector containing an internal spleen focus forming virus promoter

21 January 2018

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EphA4 (Sek1) receptor tyrosine kinase is required for the development of the corticospinal tract

21 January 2018

1 reference

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Glial cell extracellular matrix: boundaries for axon growth in development and regeneration

21 January 2018

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Eph receptors and neural plasticity

21 January 2018

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Axonal regeneration and lack of astrocytic gliosis in EphA4-deficient mice

21 January 2018

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Nogo-66 receptor antagonist peptide promotes axonal regeneration

21 January 2018

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Ascending sensory, but not other long-tract axons, regenerate into the connective tissue matrix that forms at the site of a spinal cord injury in mice

21 January 2018

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Upregulation of EphA3 receptor after spinal cord injury.

21 January 2018

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Nogo-66 receptor prevents raphespinal and rubrospinal axon regeneration and limits functional recovery from spinal cord injury

21 January 2018

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Eph/ephrin signaling in morphogenesis, neural development and plasticity.

21 January 2018

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Ephrin-B3 is the midline barrier that prevents corticospinal tract axons from recrossing, allowing for unilateral motor control

21 January 2018

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Kinase-dependent and kinase-independent functions of EphA4 receptors in major axon tract formation in vivo.

21 January 2018

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mRNA expression of ephrins and Eph receptor tyrosine kinases in the neonatal and adult mouse central nervous system

21 January 2018

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Immunolocalization of the receptor tyrosine kinase EphA4 in the adult rat central nervous system.

21 January 2018

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Corticospinal neurons up-regulate a range of growth-associated genes following intracortical, but not spinal, axotomy

21 January 2018

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A new mathematical model for relative quantification in real-time RT-PCR

21 January 2018

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Functional switch between motor tracts in the presence of the mAb IN-1 in the adult rat.

21 January 2018

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Inhibition of collagen IV deposition promotes regeneration of injured CNS axons

21 January 2018

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Changes in distribution, cell associations, and protein expression levels of NG2, neurocan, phosphacan, brevican, versican V2, and tenascin-C during acute to chronic maturation of spinal cord scar tissue

21 January 2018

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Forward signaling mediated by ephrin-B3 prevents contralateral corticospinal axons from recrossing the spinal cord midline

21 January 2018

1 reference

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Molecular basis of interactions between regenerating adult rat thalamic axons and Schwann cells in peripheral nerve grafts I. Neural cell adhesion molecules

21 January 2018

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The Eph family receptors and ligands

21 January 2018

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10.1111/J.1460-9568.2006.04704.X

21 January 2018

Identifiers

DOI

1 reference

13 February 2018

PubMed ID

16623828

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13 February 2018