Second-order fear conditioning prevented by blocking NMDA receptors in amygdala. (Q48655064)

13 February 2018

title

Second-order fear conditioning prevented by blocking NMDA receptors in amygdala. (English)

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13 February 2018

author name string

Gewirtz JC

series ordinal

1

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13 February 2018

Davis M

series ordinal

2

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13 February 2018

language of work or name

English

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publication date

1 July 1997

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13 February 2018

published in

Nature

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13 February 2018

volume

388

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13 February 2018

issue

6641

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13 February 2018

page(s)

471-474

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13 February 2018

https://scigraph.springernature.com/pub.10.1038/41325

exact match

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Equipotentiality of thalamo-amygdala and thalamo-cortico-amygdala circuits in auditory fear conditioning

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Early acquisition, but not retention, of the classically conditioned eyeblink response is N-methyl-D-aspartate (NMDA) receptor dependent.

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Spatial memory and N-methyl-D-aspartate receptor antagonists APV and MK-801: memory impairments depend on familiarity with the environment, drug dose, and training duration

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Intra-amygdala infusion of the N-methyl-D-aspartate receptor antagonist AP5 blocks acquisition but not expression of fear-potentiated startle to an auditory conditioned stimulus

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Acquisition of contextual Pavlovian fear conditioning is blocked by application of an NMDA receptor antagonist D,L-2-amino-5-phosphonovaleric acid to the basolateral amygdala

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N-methyl-D-aspartate receptors in the basolateral amygdala are required for both acquisition and expression of conditional fear in rats.

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Cocaine enhances the expression of fear-potentiated startle: evaluation of state-dependent extinction and the shock-sensitization of acoustic startle.

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Identifiers

DOI

10.1038/41325

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13 February 2018

Dimensions Publication ID

1048230555

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13 February 2018